Disease relevance of Neuritis

• Sera and buffy coat or lymph node cells from humans with idiopathic polyneuritis and animals with experimental allergic neuritis demyelinate cultures of peripheral nervous system tissue [1].
• Erythrocyte transketolase activity (ETKA) and the effect of adding thiamine pyrophosphate have been measured in a group of 27 healthy individuals and in 37 patients diagnosed as having diabetes mellitus, anemia, polyneuritis, or malnourishment secondary to vascular disease of the brain [2].
• P2 and BP antisera did not stain regions of demyelination in a case of idiopathic polyneuritis, while adjacent myelinated PN stained normally [3].
• Severe renal failure and polyneuritis induced by foscarnet [4].
• After his recovery from babesiosis, the patient developed acute idiopathic polyneuritis (Landry-Guillain-Barré Syndrome), which was probably related to his diminazene therapy [5].

High impact information on Neuritis

• Zinc pyrithione and peripheral neuritis [6].
• Expression was lower in relapsing remitting patients in remission and least in control subjects, including patients with epilepsy, chronic inflammatory demyelinating polyneuritis, and systemic lupus erythematosus [7].
• Fulminant polyneuritis after overdose of disulfiram and ethanol [8].
• Letter: Acute idiopathic polyneuritis: no residua after steroid treatment [9]?
• (f) Peripheral neuritis seen after nitrofurantoin therapy in patients with impaired renal function is thought to be due to accumulation of a toxic metabolite [10].

Chemical compound and disease context of Neuritis

• We studied the CD5 mRNA expression and VH gene family usage in Epstein-Barr virus (EBV)-immortalized B-cell lines derived from the blood of patients with type 1 diabetes (IDDM) of recent onset and of patients with polyneuritis cranialis multiplex (cranial neuritis; CN) [11].
• Clinical effects of cyclophosphamide in Guillain-Barré polyneuritis [12].
• Neuritis, disesthesias and painful sensory neuritis may resolve with good diabetic control; on occasion, diphenylhydantoin has been of therapeutic benefit [13].
• Cranial polyneuritis (Bell's palsy and Ramsay Hunt syndrome), the most common cause of facial palsy, is an inflammatory, autoimmune, demyelinating disease best treated by parenteral steroids without surgical intervention [14].
• Polyneuritis associated with azathioprine sensitivity reaction [15].

Gene context of Neuritis

• Immune complexes (IC) were assessed in serum and CSF from 11 patients with acute inflammatory polyneuritis (AIP) [16].
• No relationship could be established between CTS and the type of nephropathy, severity of polyneuritis, Ca and PO4 metabolism, the presence of vascular access and efficacy of dialysis [17].
• Neurological manifestations can very from irritability, convulsions, coma to peripheral neuritis [18].

Analytical, diagnostic and therapeutic context of Neuritis

• Since 1965, 38 patients with idiopathic polyneuritis have been at the University of Kentucky Medical Center, and 16 of them were entered in a prospective double blind study designed to assess the effectiveness of steroid therapy [19].

References