Disease relevance of MOCOS

• Psychometric variables were assessed each week by means of the West Haven-Yale Multidimensional Pain Inventory (MPI), Pittsburgh Sleep Quality Index (PSQI), Medical outcome survey-short form (MOS SF-36), the Pain Disability Index (PDI), and the Fibromyalgia Impact Questionnaire (FIQ) [1].
• Furthermore, LL, SC, LL + MOS, or SC + MOS significantly improved antibody titres against Newcastle disease virus and lymphocyte proliferation of broilers (p < 0.05) [2].

Psychiatry related information on MOCOS

• The presence of sleep disorders and the characteristics of the BDZ used were not associated with any score in the MOS SF-36 questionnaire, whereas the Prime diagnosis was the most important predictor, since subjects with depression and/or anxiety had a lower mean score on each scale than subjects without disorders [3].

High impact information on MOCOS

• Supporting these findings, recombinant C-terminal domain of the human molybdenum cofactor sulfurase exhibited N-reductive activity in vitro, which was strictly dependent on molybdenum cofactor [4].
• We assessed HRQL using the MOS 36-Item Short Form (SF-36) questionnaire, and blood pressure (BP) was measured under standardized conditions [5].
• The human molybdenum cofactor sulfurase (HMCS) gene, the human ma-l homologue, is therefore a candidate gene responsible for classical xanthinuria type II, which involves both XDH and AO deficiencies [6].
• In this study, we cloned the HMCS gene from a cDNA library prepared from liver [6].
• In two independent patients with classical xanthinuria type II, we identified a C to T base substitution at nucleotide 1255 in the HMCS gene that should cause a CGA (Arg) to TGA (Ter) nonsense substitution at codon 419 [6].

Biological context of MOCOS

• These MOSC domains contain an absolutely conserved cysteine and occur either as stand-alone forms such as the bacterial YiiM proteins, or fused to other domains such as a NifS-like catalytic domain in Molybdenum cofactor sulfurase [7].
• We evaluated and compared the two most frequently used questionnaires to assess HrQoL, namely the Nottingham Health Profile (NHP) and the MOS Short-Form-36 Health Survey (SF-36) with regard to their applicability in gerontology [8].
• The study of oculomotricity is done by the evaluation of three systems: saccade eye movements (MOS), optokinetic nystagmus (NO) and smooth pursuit eye movement (MORL) [9].

Associations of MOCOS with chemical compounds

• The chromatography was performed on a MOS Hypersil (C(8)) column and was optimized by investigating the effects of methanol (7.5-20%), acetonitrile (5-7%), tetrabutylammonium hydrogen-sulphate (TBA, 0-90 mM) and acetate buffer (pH 3.5, 60-100 mM) on the retention of fetindomide and mitindomide [10].
• Health-related quality of life was measured using the MOS 36-item Short Form Health Survey (SF-36), and the US Centers for Disease Control healthy day's items [11].
• However, compared to the control, LL, SC, LL + MOS, or SC + MOS significantly decreased malondialdehyde concentration in serum, thigh, and heart of broilers [2].

Analytical, diagnostic and therapeutic context of MOCOS

• HRQoL was measured by the eight MOS 36-Item Short-Form Health Survey (SF-36) scales [12].

References