Disease relevance of Open Bite

• Skeletal anterior open bite, overjets greater than 6 to 7 mm, retruded cuspal position/intercuspal position slides greater than 4 mm, unilateral lingual crossbite, and five or more missing posterior teeth are the five occlusal features that have been associated with specific diagnostic groups of TMD conditions [1].
• The oral findings comprise micrognathia, hypodontia, localized microdontia, opalescent tooth discoloration, root dysplasia, pulp obliteration, severe gingival hyperplasia, frontal open bite, and severe restriction of TMJ mobility [2].
• It is concluded that that the prevalence of temporomandibular disorders in anterior open bite patients increases with age, is significantly higher in females, and is not influenced by other occlusal variables [3].
• In addition to a pathology literature review, this article describes an unusual case of temporomandibular synovial chondromatosis that was sufficiently expansile to displace the condyle, which created not only a posterior occlusal apertognathia, but unusual articular bony fossa and eminentia erosions [4].
• Activity in temporalis and masseter muscles, and traits of facial morphology and occlusal stability were studied in 22 patients (19 women, 3 men; 15-45 yr of age) with anterior open bite and symptoms and signs of craniomandibular disorders [5].

High impact information on Open Bite

• The adult open-bite group showed a weaker occlusal force and a shorter range of condylar motion compared with the control subjects [6].
• Peptide histidine methionine induced a significant increase in the maximal CCP obtained in nonocclusion, and the time interval until return of CCP to steady state levels was increased in both aortic nonocclusion and occlusion [7].
• The clinical examination included registration of decayed, extracted and filled teeth and surfaces (deft and defs), gingival bleeding points (GBI) and presence of open bite or crossbite [8].
• BACKGROUND: Mandible advancement osteotomy in juvenile rheumatoid arthritis patients for class II anterior open-bite malocclusion is controversial; however, there is no literature discussing outcomes of surgery in this group of patients [9].
• The study group comprised 16 consecutively treated open-bite cases who had received special titanium miniplates in their zygomatic bones for use as anchorage to apply orthodontic intrusive forces to the maxillary posterior region [10].

Chemical compound and disease context of Open Bite

• This article reports the successful treatment of a severe skeletal anterior open-bite case using titanium screw anchorage [11].
• Steroid-induced anterior open bite. Case report [12].
• This investigation examined the magnetic flux and repulsive force of neodymium-iron-boron (Nd-Fe-B) magnets used in repulsion in the treatment of anterior open bite [13].
• After 5 minutes of occlusion (Orahesive Oral Bandages, ConvaTec) with a eutectic mixture of local anesthetics (EMLA cream, Astra), nociceptive pinprick thresholds on the tongue and gingiva are higher than those taken during nonocclusion [14].

Biological context of Open Bite

• It is suggested that the frequent association of anterior open-bite and amelogenesis imperfecta is caused by a genetically determined anomaly of craniofacial development, rather than by local factors influencing alveolar growth [15].
• The phenotype associated with the g.13185_13186insAG ENAM mutation is dose dependent such that ARAI with openbite malocclusion segregates as a recessive trait, and enamel pitting as a dominant trait [16].

Anatomical context of Open Bite

• Our results suggest that titanium screws are useful for intrusion of molars in anterior open-bite cases [11].
• Sleep apnea subjects showed a posteriorly positioned maxilla and mandible, a steep occlusal plane, overerupted maxillary and mandibular teeth, proclined incisors, a steep mandibular plane, a large gonial angle, high upper and lower facial heights, and an anterior open bite in association with a long tongue and a posteriorly placed pharyngeal wall [17].
• Anterior open bite as a clinical sign of joint destruction was found to be associated with high levels of NPY and IL-1beta in the synovial fluid [18].
• It was found that, Class III malocclusion in the permanent dentition and openbite, overbite = 0, overjet = 0, anterior-posterior crossbite in the mixed dentition were related with TMD [19].

Gene context of Open Bite

• Nine adult open bite patients (7 women and 2 men) successfully treated with SAS were included in the following study [20].
• Mediotrusion interferences were more consistent with basal morphological deviations, for example, Class III relationships and anterior open bite were more consistent in the same person, and more difficult to eliminate than RCP/ICP interferences [21].
• Totally asymptomatic controls were characterized by a lack of anterior open bite, small symmetric RCP-ICP slides (greater than 0 less than 1 mm), and bilateral occlusal contact in RCP [22].
• Maxillary overjet accounted for 13%, deep bite 13%, dental midline displacement 6%, frontal openbite 12% and anterior crossbite 5% [23].
• Orthodontic treatment of an open bite patient with oral-facial-digital syndrome [24].

Analytical, diagnostic and therapeutic context of Open Bite

• Occlusal therapy to correct apertognathia after mandibular osteotomy [25].

References