Disease relevance of Host-Parasite Relations

• Effect of cortisone administration on host-parasite relationships in early experimental syphilis [1].
• Homozygous mutant scid/scid (severe combined immunodeficiency) mice (referred to as scid mice) lack both specific humoral and cell-mediated immune functions and are exemplary in vivo models for analysis of host-parasite relationships [2].
• Filariae also appear to require other specific nutrients such as iodine, thiamine and pyridoxine; dietary levels of these nutrients affect the host-parasite relationships in filariasis and pyridoxine seems to be of particular importance in this respect [3].
• The significant and distinctive regional changes of c-fos in the CNS of infected mice indicate that the brain of the host senses intraperitoneal cysticercosis and may also announce its active participation in the regulation of the host-parasite relationship [4].
• The enzyme is an immunodominant antigen in human chronic Chagas disease and seems to be important in the host/parasite relationship [5].
• Host-parasite relationships depends on genotype of the host. One of the well studied models of these interactions is Leishmania major infection in mouse (reviewed in [6].
• Genetic studies revealed distinct genetic control of parasite elimination, dissemination, and disease after Leishmania major infection [7].

Psychiatry related information on Host-Parasite Relations

• Finally, these data also supply evidence for the evolutionary conservation of the function and role of interleukin-1, O2-, and antioxidant defense mechanisms in this host-parasite relationship [8].

High impact information on Host-Parasite Relations

• The interaction of SP-D with gpA represents an additional important component of the host-parasite relationship during P. carinii pneumonia [9].
• Taken together, these studies indicate that P. gingivalis arginine-specific protease can expose a cryptitope in the matrix protein molecules, i.e. the C-terminal Arg residue of the host matrix proteins, so that the organism can adhere to the surface layer in the oral cavity through fimbriae-Arg interaction (a novel host-parasite relationship) [10].
• The pentose phosphate pathway plays a crucial role in the host-parasite relationship [11].
• The results of this study have important implications for the role of IL-7 in the host-parasite relationship and show how parasites can disable or evade the host immune response [12].
• We anticipate that the kininogen-M protein interaction contributes to the host-parasite relationship in S. pyogenes infections [13].

Biological context of Host-Parasite Relations

• Knowing the nature of the mycobacterial constituents that induce iNOS gene expression would help to better understand the host-parasite relationship [14].
• The present data represent a step forward into the understanding of the signal transduction process in the host-parasite relationship by supporting the idea that the host hormone melatonin and N-acetyl-serotonin generate IP3 and therefore mobilize intracellular Ca2+ in Plasmodium inside red blood cells [15].

Anatomical context of Host-Parasite Relations

• The implications of these activations on the host-parasite relationship are discussed and compared with LPS-stimulated macrophages [16].

Associations of Host-Parasite Relations with chemical compounds

• These data suggest that heparin may play a role in the host-parasite relationship [17].
• The host-parasite relationship in taeniosis due to Taenia solium is practically unknown [18].
• Since S. mansoni paramyosin is thought to be a muscle protein and T. solium antigen B a secreted glycoprotein with anti-complement activity, this conclusion raises some interesting questions regarding the role of this class of proteins in the host-parasite relationship [19].
• The host-parasite relationships in pyridoxine (vitamin B6) deficient cotton rats infected with Litomosoides carinii (Nematoda: Filaroidea) [20].
• Cyclosporin A: antiparasite drug, modulator of the host-parasite relationship and immunosuppressant [21].
• First line drugs, alternative drugs and experimental treatment of leishmaniasis is decribed in [22].

Gene context of Host-Parasite Relations

• The major lysosomal cysteine proteinase, cruzipain, has been characterized in considerable detail, and might be involved in the host/parasite relationship, in addition to its obvious role in parasite nutrition [23].
• Prospective roles for hookworm calreticulin in the host-parasite relationship are discussed in depth [24].
• The results show the potential of this technique for detecting proteinases in trichomonad samples in studies aimed at determining proteinase function in pathogenesis and host-parasite relationships [25].
• The importance of inducible immunoregulatory networks in the ecology of the host-parasite relationship is considered, with particular regard to possible manipulative strategies by the parasites [26].
• Schistosoma mansoni host-parasite relationship: interaction of contrapsin with adult worms [27].

Analytical, diagnostic and therapeutic context of Host-Parasite Relations

• Platelet TLX offers a new genetic and immunological approach to study similarities of the host-parasite relationships in pregnancy, transplantation, and cancer [28].
• The neuropathology and host-parasite relationship of experimental infection with the RH-strain of Toxoplasma gondii were studied in 27 blue foxes (Alopex lagopus) aged 0 to 23 days, using light microscopy, including immunohistochemical staining, and transmission and scanning electron microscopy [29].

References