Disease relevance of Sinus Thrombosis, Intracranial

• Dural sinus thrombosis developed in two children with acute lymphoblastic leukemia during induction treatment with vincristine sulfate, prednisone, and asparaginase [1].
• Activated protein C resistance and factor V Leiden mutation are risk factors for cerebral sinus thrombosis [2].
• Six women had free protein S antigen levels below the range recorded for a contemporary group of 24 age-matched normal women (17 to 59% versus 70 to 102%, p less than 0.001); four of these women had cerebral arterial thrombosis and two had venous dural sinus thrombosis [3].
• Rheolytic catheter thrombectomy, balloon angioplasty, and direct recombinant tissue plasminogen activator thrombolysis of dural sinus thrombosis with preexisting hemorrhagic infarctions [4].
• Dural sinus thrombosis with severe hypernatremia developing in a patient on long-term lithium therapy [5].

High impact information on Sinus Thrombosis, Intracranial

• Increased risk of cerebral venous sinus thrombosis with third-generation oral contraceptives. Cerebral Venous Sinus Thrombosis Study Group [6].
• Heparin treatment for cerebral venous and sinus thrombosis [7].
• Cerebral sinus thrombosis with tamoxifen [8].
• Because androgen use may be frequent and hidden in athletes, it may be an underestimated cause of cerebral venous sinus thrombosis in the young [9].
• Although increased risk of thromboembolic disease has been reported during treatment with epsilon-aminocaproic acid, cerebral sinus thrombosis has not been previously described [10].

Chemical compound and disease context of Sinus Thrombosis, Intracranial

• Gadolinium-enhanced MR of chronic dural sinus thrombosis [11].
• The sinus thrombosis was considered to have been a side effect of the medroxyprogesterone acetate medication [12].
• Gadopentetate dimeglumine-enhanced MR imaging was performed in four patients with chronic dual sinus thrombosis [11].
• Venous sinus thrombosis in a patient taking thalidomide [13].
• Gadolinium-DTPA enhanced MR imaging of septic dural sinus thrombosis [14].

Anatomical context of Sinus Thrombosis, Intracranial

• Mastoiditis, meningitis, petrositis, sigmoid sinus thrombosis, extradural abscess, brain abscess, otitic hydrocephalus, cholesteatoma and cholesterol granuloma are complications of otitis media and middle ear disease [15].

Gene context of Sinus Thrombosis, Intracranial

• Direct endovascular thrombolytic therapy for dural sinus thrombosis: infusion of alteplase [16].
• Plasminogen activator inhibitor-1 4G/4G-genotype is associated with cerebral sinus thrombosis in factor V Leiden carriers [17].
• CLINICAL PRESENTATION: Three patients developed large hemorrhagic infarct with coma and bilaterally fixed and dilated pupils resulting from aseptic dural sinus thrombosis [18].
• Dural sinus thrombosis in childhood systemic lupus erythematosus [19].
• Dural sinus thrombosis associated with activated protein C resistance: MR imaging findings and proband identification [20].

Analytical, diagnostic and therapeutic context of Sinus Thrombosis, Intracranial

• Case-control study of risk of cerebral sinus thrombosis in oral contraceptive users and in [correction of who are] carriers of hereditary prothrombotic conditions. The Cerebral Venous Sinus Thrombosis Study Group [21].

References