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MeSH Review

Electric Countershock

 
 
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Disease relevance of Electric Countershock

 

Psychiatry related information on Electric Countershock

 

High impact information on Electric Countershock

 

Chemical compound and disease context of Electric Countershock

 

Biological context of Electric Countershock

 

Anatomical context of Electric Countershock

  • The present studies performed in experimental animals demonstrate that electrical direct current cardioversion can produce skeletal muscle damage and increased technetium-99m stannous pyrophosphate (99mTc-PYP) uptake; in experimental animals the electrically damaged skeletal muscle shows necrosis with extensive calcium deposition [20].
  • CONCLUSIONS: In patients with persistent AF, long-term administration of verapamil before internal cardioversion resulted in 1) shortening of atrial ERPs; 2) no change in refractoriness dispersion within the right atrium; and 3) no change in atrial ERP adaptation to rate [21].
  • Thus, short-term amiodarone with or without electrical cardioversion is effective and safe in the treatment of chronic rheumatic AF after mitral valve surgery [22].
  • RESULTS: With infusion of 0.005-0.008 microg/kg/min isoproterenol, heart rate increased by 11.1+/-2.9%, and left atrial appendage emptying velocity, which was diminished following cardioversion, increased significantly (P<0.001) (baseline, before and after isoproterenol: 41.1+/-18.0, 20.3+/-8.5 and 27.3+/-9.6 cm/s, respectively) [23].
  • AIM OF THE STUDY: To evaluate the effectiveness of Ibutilide in cardioversion of persistent atrial fibrillation in patients with sinus node disease wearing a dual chamber pacemaker and to assess the potential role of overdrive ventricular pacing in prevention of drug related proarrhythmia [24].
 

Associations of Electric Countershock with chemical compounds

 

Gene context of Electric Countershock

  • Baseline cortisol levels, however, were significantly higher in the depressed group compared with the cardioversion group, P < 0.02, but not ACTH or AVP [27].
  • There was no significant difference in CRH responses between cardioversion and control days [27].
  • CONCLUSION: Significant hypothalamic-pituitary-adrenal activation and PRL release occur in response to both cardioversion and ECT [27].
  • Levels of hs-CRP, IL-6, and TNF-alpha after cardioversion were significantly higher than those in controls (P < 0.05) [28].
  • CONCLUSION: A strategy of approximately 24-hour delay in cardioversion shock timing decreased the incidence of ERAF, relative to a shock delivered within a few hours of AF onset [29].
 

Analytical, diagnostic and therapeutic context of Electric Countershock

References

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  26. Efficacy and safety of quinidine therapy for maintenance of sinus rhythm after cardioversion. A meta-analysis of randomized control trials. Coplen, S.E., Antman, E.M., Berlin, J.A., Hewitt, P., Chalmers, T.C. Circulation (1990) [Pubmed]
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