Disease relevance of Persian Orange

• Streptomyces spp. decolorized Orange I but not Acid Orange 12 or Orange II [1].
• The blend of Metanil yellow and Orange II (40 mg + 40 mg/kg body weight) given intraperitoneally for 3 days resulted in either synergistic or additive effect in nuclear (122%), mitochondrial (130%), and microsomal (62%) membranes [2].
• Properties of purified Orange II azoreductase, the enzyme initiating azo dye degradation by Pseudomonas KF46 [3].
• Two sulfonated azo dyes, Orange II and Amido black, are effectively decolorized by certain nocardioform strains of the genus Rhodococcus [4].
• Acute and short-term toxicity studies on orange II [5].

High impact information on Persian Orange

• Photobleaching of Orange II within seconds using the oxone/Co2+ reagent through Fenton-like chemistry [6].
• Azo1 was also able to metabolize Orange II, Amaranth, Ponceau BS and Ponceau S azo dyes [7].
• The apparent discoloration kinetics of Orange II with the four catalysts at two different initial solution pH values was also investigated [8].
• The reaction activation energy calculated was 9.94 kJ/mol, indicating that the photo-Fenton discoloration of Orange II is not very sensitive to reaction temperature [9].
• The same dose of Orange II to rats showed a relatively less but significant effect in nuclear (62%), mitochondrial (59%) and microsomal (38%) membranes [2].

Chemical compound and disease context of Persian Orange

• Orange II azoreductase [NAD(P)H: 1-(4'-sulfophenylazo)-2-naphthol oxidoreductase], an enzyme catalyzing the reductive cleavage of the azo bridge of Orange II and related dyes, was purified to electrophoretic homogeneity from Pseudomonas species, strain KF46 [3].
• Parenteral administration of Metanil yellow and Orange II to rats at a dose of 80 mg/kg body weight for 3 days caused a significant induction of ethoxyresorufin-O-deethylase (40-190%), aniline hydroxylase (27-92%), aryl hydrocarbon hydroxylase (50-62%) and aminopyrine N-demethylase (42-49%) activities [10].

Biological context of Persian Orange

• Fresnel lens to concentrate solar energy for the photocatalytic decoloration and mineralization of orange II in aqueous solution [11].
• Oral LD50 value of orange II in both male and female rats was calculated to be more than 10.56 g/kg body weight [5].
• The reductive biotransformation of acid orange 7 (AO7) was explored in a lab-scale upflow anaerobic sludge blanket (UASB) reactor at low hydraulic residence times (HRT) [12].
• Orange II, C.I. Acid Orange 7 (AO7), is oxidatively decolorized via catalytic oxidation by iron(III) phthalocyanine-tetrasulfonic acid (Fe(III)-PcTS) as a biomimetic catalyst and KHSO(5) as an oxygen donor [13].
• The water purification potential of this new class of compound was evaluated by studying the photodegradation of Acid Orange 7 (AO7) and E. coli [14].

Anatomical context of Persian Orange

• Kinetic response of testis to Orange II administration in rat [15].

Associations of Persian Orange with other chemical compounds

• The decolorization of C.I. Acid Orange 7 (AO7), an anionic monoazo dye of acid class, was investigated using UV radiation in the presence of H2O2 in a tubular continuous-flow photoreactor as a function of oxidant and dye concentrations, reactor length and volumetric flow rate [16].
• This study examined the photocatalytic degradation of three azo dyes, acid orange 7 (AO7), procion red MX-5B (MX-5B) and reactive black 5 (RB5) using a new type of nitrogen-doped TiO2 nanocrystals [17].
• The amount of H(2)O(2) and the pH was optimized for the photobleaching of Orange II on the H(5)FeW(12)O(40)/silica fabric [18].
• Metanil yellow and Orange II brought about a substantial increase in cytosolic quinone reductase (34-82%) and glutathione S-transferase (23-43%) activities and significant depletion of glutathione levels with a concomitant increase in lipid peroxide formation [10].
• Kinetics of anaerobic biodecolourisation (methanogenic environment) of four azo dyes (Acid Orange 6, Acid Orange 7, Methyl Orange and Methyl Red) was investigated with regard to their electrochemical properties as well as under variation of dye and sludge concentrations, pH and temperature [19].

Gene context of Persian Orange

• The photodestruction of Acid Orange 7 (AO7), an anionic acidic dye, was studied in the UV/H2O2 process [20].
• Under optimal conditions (pH=3.0, 10 mM H(2)O(2), and 1 x 8W UVC), 100% discoloration and 50-60% TOC removal of 0.2 mM Orange II can be achieved in 90 and 120 min, respectively [21].
• The sorption of artificial dye effluent made from two different dyes, Solar orange 7 GLL and Solar Jade Green FFB from Clariant, onto three different agricultural residues--barley husk, sugarcane bagasse, and wheat straw--was studied [22].
• Orange II induced cytogenetical changes in albino mice [23].
• The adsorption capacity was pH dependent with a maximum value of 109.0 mg/g at pH 7 for AR88, 133.5 mg/g at pH 3 for AG3 and 109.6 mg/g at pH 3 for AO7, respectively, was obtained [24].

Analytical, diagnostic and therapeutic context of Persian Orange

• For the gelatin-free microspheres, a close correlation of SA release and AO release was observed (2% loading), suggesting a release mechanism that was controlled dominantly by degradation [25].
• A packed-bed fungal bioreactor for the continuous decolourisation of azo-dyes (Orange II) [26].
• Experiments above demonstrate that TiO(2) modified beta-PbO(2) electrode, which realized TiO(2) immobilization successfully, performed well in EAPD of Acid Orange 7 [27].
• The adsorption capacities of the derived adsorbents for Acid Orange 10 were measured at 20 degrees C and 40 degrees C to gain further insights into the acidic surface oxides of the adsorbent from the results of Fourier transform infrared (FTIR) spectroscopy analysis and pH measurement [28].
• The influence of a toxic organic compound (acid orange 7) on biofilm microprofiles was also monitored using microelectrodes [29].

References