Disease relevance of Bottle Feeding

• Liver disease in alpha 1-antitrypsin deficiency. A retrospective analysis of the influence of early breast- vs bottle-feeding [1].
• METHODS: To evaluate the risk of hypoglycemia in low grade low-birthweight infants (LGLBWI) (2100 g < birthweight < 2500 g) whose only abnormality is low-birthweight, whole blood glucose (BGw) was measured five times (0, 0.5, 1, and 4 h after birth and just before the first bottle feeding) in 49 LGLBWI and 38 normal birthweight infants [2].
• Now Mitchell (New Zealand Herald, 27.11.90) has concluded a 3-year study of SIDS' victims in New Zealand and found a combination of stomach sleeping position, mothers' smoking and bottle feeding was implicated in 79% of SIDS' victims [3].

High impact information on Bottle Feeding

• He roused from his coma, breathed spontaneously, and resumed bottle feeding [4].
• In 1978, a retrospective study of the influence of sociodemographic factors on the trend in breast- and bottle-feeding was conducted among a sample of Pima Indian women 15 to 44 yr old residing on the Gila River Reservation [5].
• Under double blind conditions 55 young, bottle feeding, multiparous women received L-tryptophan 3 g daily or placebo for ten days after delivery, and were also assessed daily for psychiatric symptoms [6].
• RESULTS: Circulating levels of PTHrP were readily detectable in 12 of 19 nursing mothers (range 2.7-7.8 pmol/l) but in none of the mothers who were bottle feeding [7].
• In an open pilot study with a parallel group design 30 bottle feeding women were randomly assigned in a two to one ratio to receive either the new dopamine agonist CV 205-502 or bromocriptine for lactation inhibition [8].

Biological context of Bottle Feeding

• These included: water fluoride concentration, breast-feeding duration, professionally applied topical fluoride, fluoride supplement use, bottle-feeding, fluoridated mouthwash use, and early parental toothbrushing with toothpaste [9].
• Energy expenditure at rest and in response to a meal and to an infusion of noradrenaline was measured in 12 lactating women and compared with that in seven bottle feeding women and seven non-pregnant, non-lactating controls [10].
• The effects of NPO, nasogastric tubes, different nipples, breast versus bottle feeding, gestational age, oral support, oral stimulation, and nonnutritive sucking on volume intake are evaluated [11].

Anatomical context of Bottle Feeding

• Flow and pressure in the thoracic aorta and pressure in the distal abdominal aorta were measured under control conditions and during pressure changes caused by bottle feeding or during intravenous infusions of nitroprusside, norepinephrine, or angiotensin II [12].

Associations of Bottle Feeding with chemical compounds

• The Pima infant feeding study: the role of sociodemographic factors in the trend in breast- and bottle-feeding [5].
• Both lithium and diazepam have been reported to cause deleterious side effects on breast-fed infants, and as the side effects of other psychotropic drugs given to a nursing mother are imperfectly understood, bottle feeding seems prudent [13].
• A 0.1% tartrate or a 0.25% saline solution was given to each infant after 1-2 min of breast or bottle feeding and then the same solutions were once more given to the same subjects [14].
• Multiple regression analyses revealed that variables associated with efficient infant performance included higher birthweight, less need of ventilator and oxygen treatment, higher haemoglobin level, absence of bottle-feeding, no need of apnoea treatment with Theophylline, and no suspicion of infection [15].
• The discussion of this crucial aspect of care includes the implications of physiologic immaturity and current recommendations for intervention as infants progress from intravenous fluids and electrolytes to parenteral nutrition, gavage feeding, and bottle feeding [16].

Gene context of Bottle Feeding

• The study supports the following conclusions: (a) neonatal hypergastrinemia is mainly due to G34 fraction; (b) the increased levels of gastrin on the 4th day of life are due to G17 fraction; (c) bottle-feeding does not stimulate either G17 or G34 release in the first 4 days of life [17].
• Logistic regression analysis revealed that positive symptom status related significantly to EA and CVS interventions (P < 0.0001), bottle feeding (P < 0.001), parental smoking (P < 0.01), a family history of atopy (P < 0.01), and immaturity (P < 0.01) [18].
• Each of the two groups was fed with either ready-to-use formula or with reconstructed powdered formula (using a two-compartment patented feeding bottle called Twist 'N Feed) and one group received both preparations during 2 successive days [19].
• Instruments in neonatal research: measuring preterm infant feeding ability, Part I: Bottle feeding [20].
• Thus, the relationship of OT responsivity to BP in 14 breast- and 11 bottle-feeding mothers of infants was examined [21].

References