Gene Review:
Casq1 - calsequestrin 1
Mus musculus
Synonyms:
CSQ, CSQ-1, CSQ1, Calmitine, Calsequestrin, skeletal muscle isoform, ...
- Regulation of Ca2+ signaling in transgenic mouse cardiac myocytes overexpressing calsequestrin. Jones, L.R., Suzuki, Y.J., Wang, W., Kobayashi, Y.M., Ramesh, V., Franzini-Armstrong, C., Cleemann, L., Morad, M. J. Clin. Invest. (1998)
- Rescue of contractile parameters and myocyte hypertrophy in calsequestrin overexpressing myocardium by phospholamban ablation. Sato, Y., Kiriazis, H., Yatani, A., Schmidt, A.G., Hahn, H., Ferguson, D.G., Sako, H., Mitarai, S., Honda, R., Mesnard-Rouiller, L., Frank, K.F., Beyermann, B., Wu, G., Fujimori, K., Dorn, G.W., Kranias, E.G. J. Biol. Chem. (2001)
- Remodelling of ionic currents in hypertrophied and failing hearts of transgenic mice overexpressing calsequestrin. Knollmann, B.C., Knollmann-Ritschel, B.E., Weissman, N.J., Jones, L.R., Morad, M. J. Physiol. (Lond.) (2000)
- Cardiac-specific overexpression of calsequestrin results in left ventricular hypertrophy, depressed force-frequency relation and pulsus alternans in vivo. Schmidt, A.G., Kadambi, V.J., Ball, N., Sato, Y., Walsh, R.A., Kranias, E.G., Hoit, B.D. J. Mol. Cell. Cardiol. (2000)
- Changes in calcium cycling precede cardiac dysfunction during autoimmune myocarditis in mice. Stull, L.B., Matteo, R.G., Sweet, W.E., Damron, D.S., Schomisch Moravec, C. J. Mol. Cell. Cardiol. (2001)
- The L-type calcium channel inhibitor diltiazem prevents cardiomyopathy in a mouse model. Semsarian, C., Ahmad, I., Giewat, M., Georgakopoulos, D., Schmitt, J.P., McConnell, B.K., Reiken, S., Mende, U., Marks, A.R., Kass, D.A., Seidman, C.E., Seidman, J.G. J. Clin. Invest. (2002)
- A transgenic myogenic cell line lacking ryanodine receptor protein for homologous expression studies: reconstitution of Ry1R protein and function. Moore, R.A., Nguyen, H., Galceran, J., Pessah, I.N., Allen, P.D. J. Cell Biol. (1998)
- Defective beta-adrenergic receptor signaling precedes the development of dilated cardiomyopathy in transgenic mice with calsequestrin overexpression. Cho, M.C., Rapacciuolo, A., Koch, W.J., Kobayashi, Y., Jones, L.R., Rockman, H.A. J. Biol. Chem. (1999)
- Cloning of the genes encoding mouse cardiac and skeletal calsequestrins: expression pattern during embryogenesis. Park, K.W., Goo, J.H., Chung, H.S., Kim, H., Kim, D.H., Park, W.J. Gene (1998)
- Regulation of GATA-4 and AP-1 in transgenic mice overexpressing cardiac calsequestrin. Suzuki, Y.J., Ikeda, T., Shi, S.S., Kitta, K., Kobayashi, Y.M., Morad, M., Jones, L.R., Blumberg, J.B. Cell Calcium (1999)
- Cardiac-specific overexpression of mouse cardiac calsequestrin is associated with depressed cardiovascular function and hypertrophy in transgenic mice. Sato, Y., Ferguson, D.G., Sako, H., Dorn, G.W., Kadambi, V.J., Yatani, A., Hoit, B.D., Walsh, R.A., Kranias, E.G. J. Biol. Chem. (1998)
- Calsequestrin, a calcium sequestering protein localized at the sarcoplasmic reticulum, is not essential for body-wall muscle function in Caenorhabditis elegans. Cho, J.H., Oh, Y.S., Park, K.W., Yu, J., Choi, K.Y., Shin, J.Y., Kim, D.H., Park, W.J., Hamada, T., Kagawa, H., Maryon, E.B., Bandyopadhyay, J., Ahnn, J. J. Cell. Sci. (2000)
- Vesicle budding from endoplasmic reticulum is involved in calsequestrin routing to sarcoplasmic reticulum of skeletal muscles. Nori, A., Bortoloso, E., Frasson, F., Valle, G., Volpe, P. Biochem. J. (2004)
- A retrograde signal from calsequestrin for the regulation of store-operated Ca2+ entry in skeletal muscle. Shin, D.W., Pan, Z., Kim, E.K., Lee, J.M., Bhat, M.B., Parness, J., Kim, d.o. .H., Ma, J. J. Biol. Chem. (2003)
- Localization and characterization of the calsequestrin-binding domain of triadin 1. Evidence for a charged beta-strand in mediating the protein-protein interaction. Kobayashi, Y.M., Alseikhan, B.A., Jones, L.R. J. Biol. Chem. (2000)
- Targeted inhibition of beta-adrenergic receptor kinase-1-associated phosphoinositide-3 kinase activity preserves beta-adrenergic receptor signaling and prolongs survival in heart failure induced by calsequestrin overexpression. Perrino, C., Naga Prasad, S.V., Patel, M., Wolf, M.J., Rockman, H.A. J. Am. Coll. Cardiol. (2005)
- Junctin and calsequestrin overexpression in cardiac muscle: the role of junctin and the synthetic and delivery pathways for the two proteins. Tijskens, P., Jones, L.R., Franzini-Armstrong, C. J. Mol. Cell. Cardiol. (2003)
- Surface plasmon resonance studies prove the interaction of skeletal muscle sarcoplasmic reticular Ca(2+) release channel/ryanodine receptor with calsequestrin. Herzog, A., Szegedi, C., Jona, I., Herberg, F.W., Varsanyi, M. FEBS Lett. (2000)
- Tubular aggregates are from whole sarcoplasmic reticulum origin: alterations in calcium binding protein expression in mouse skeletal muscle during aging. Chevessier, F., Marty, I., Paturneau-Jouas, M., Hantaï, D., Verdière-Sahuqué, M. Neuromuscul. Disord. (2004)