Disease relevance of Tachycardia, Sinus

• Programmed stimulation could not induce the tachycardia but high dose propranolol therapy failed to suppress sinus tachycardia in response to isoproterenol infusion [1].
• Thus falipamil can be used to normalize sinus rate in patients with sinus tachycardia of various origin, particularly in intensive care, in acute ischaemic heart disease, in anaesthesia and in surgery cases [2].
• RESULTS: (1) Thyroxine resulted in 20% cardiac hypertrophy, with normal arterial tension, sinus tachycardia, a shorter P wave length and PR interval, and frequent (5/6) atrioventricular block [3].
• A plasma hydroxyzine concentration 8.5 hours after the acute ingestion was 102.7 micrograms/ml and toxicity was manifested primarily by generalized seizures and sinus tachycardia [4].
• Diltiazem to treat sinus tachycardia in critically ill patients: a four-year experience [5].

Psychiatry related information on Tachycardia, Sinus

• Symptoms of pemoline ingestion appear to be primarily an accentuation of the drug's pharmacological effects on the central nervous and cardiovascular systems with sinus tachycardia, hypertension, hyperactivity, choreoathetoid movements, and hallucinations being most commonly observed [6].
• We evaluated the clinical effectiveness of esmolol, an ultra-short-acting beta 1-adrenergic receptor blocking drug, to control the sinus tachycardia and increase in arterial blood pressures induced by electroconvulsive therapy (ECT) [7].

High impact information on Tachycardia, Sinus

• We hypothesized that the sinus tachycardia of exercise may enhance flecainide-induced conduction slowing by increasing use-dependent sodium channel blockade, thereby facilitating the occurrence of ventricular reentry [8].
• Salbutamol injections caused a sinus tachycardia [9].
• BACKGROUND: Transient superior vena cava occlusion has been reported following catheter ablation procedures for inappropriate sinus tachycardia (IST) [10].
• In 20 patients with high plasma norepinephrine concentrations within the first 24 hours, sinus tachycardia (p less than 0.02) and negative T waves (p less than 0.01) were more frequent than in the 20 patients with normal plasma norepinephrine concentrations [11].
• Atropine-induced and catecholamine-induced sinus tachycardia is also significantly diminished [2].

Chemical compound and disease context of Tachycardia, Sinus

• After 72 hours, only sinus tachycardia (p less than 0.03) was found with increased frequency in the 26 patients with high plasma norepinephrine concentrations [11].
• The haemodynamic response to alinidine warrants further studies of its efficacy in patients with angina or sinus tachycardia [12].
• Occlusion of the sinus node artery induced a sinus tachycardia which was not blocked by sotalol [13].
• At higher dosages, patients receiving levosimendan had higher rates of sinus tachycardia than those in placebo recipients [14].
• Sphingosine 1-phosphate induces sinus tachycardia and coronary vasoconstriction in the canine heart [15].

Biological context of Tachycardia, Sinus

• 4. All the animals developed sinus tachycardia (above 200 beats min-1) within 30 s of each dose of salbutamol administration and the high heart rate persisted throughout the experiment [16].
• We evaluated the clinical effectiveness of esmolol, an ultra-short-acting, cardioselective beta-adrenergic receptor blocker, in controlling sinus tachycardia and increased systolic blood pressure occurring perioperatively in 30 ASA physical status II or III patients having elective, non-cardiac surgery [17].
• Cardiotoxicity occurred in 20% of the patients after 14-30 mg/m2 mitoxantrone; these included complete heart block with fatal outcome in one case, decreased ventricular ejection fraction in one, and sinus tachycardia in two [18].

Anatomical context of Tachycardia, Sinus

• The results demonstrated that comparable PDI values and PDI changes with epinephrine induced sinus tachycardia were obtained with all three tested sensing configurations: conventional unipolar, tip electrode to right ventricular defibrillation electrode, and tip electrode to superior vena cava defibrillation electrode [19].
• The findings suggest that (1) there is a high incidence of supraventricular arrhythmias and sinus tachycardia after coronary artery bypass which might reflect an abrupt propranolol withdrawal, and (2) that perioperative prophylactic or supplementary propranolol therapy will successfully prevent or treat most of these arrhythmias [20].
• In the tetrodotoxin treated preparation in which the effect of electrical stimulation of the vagus and the stellate ganglion was abolished, tyramine produced an increase of sinus rate as usual, but the induction of sinus tachycardia by cocaine and ketamine was completely blocked [21].

Gene context of Tachycardia, Sinus

• Our results suggest that inhibition of both types of GIRK channels by these drugs underlies some of the side effects, in particular seizures and sinus tachycardia, observed in clinical practice [22].
• Noninvasive discrimination of right atrial ectopic tachycardia from sinus tachycardia in "dilated cardiomyopathy" [23].
• Masseter spasm and sinus tachycardia were as common in MHS as in MHN individuals [24].
• An adaptive interval-based algorithm for withholding ICD therapy during sinus tachycardia [25].
• Typical initial manifestations of CI in DMD/BMD are sinus tachycardia, tall R1 in V1, prominent Q in I, aVL, V6 or in II, III, and aVF, increased QT dispersion and possibly autonomic dysfunction [26].

Analytical, diagnostic and therapeutic context of Tachycardia, Sinus

• Beta-receptor blockade was achieved by selective perfusion of propranolol hydrochloride solution (10 mug/ml) into the sinus node artery and did not prevent the initial sinus tachycardia [27].
• ECG demonstrated sinus tachycardia with poor R wave progression in precordial leads and T wave inversion in leads V1-3 [28].

References