Disease relevance of Food Preferences

• Decreased SWS and MIC consumption in RYGB patients suggests that food-preference differences are partially responsible for the lower calorie intake and greater weight loss after RYGB than after HGP [1].
• During use and/or withdrawal from nicotine, heroin, marijuana, and cocaine, major changes in food selection and intake occur, which result in weight gain or loss [2].
• Clinical signs commonly attributed to malocclusion, such as ventral mandibular swelling, weight loss, dysphagia, altered chewing pattern and changed food preferences, were not specific to malocclusion, being seen associated with coronal spike formation, root elongation and advanced periodontal lesions [3].

Psychiatry related information on Food Preferences

• Neonatal lesions were induced with ibotenate on postnatal day (pd) 7 and all animals were tested pre- and postpubertally for prepulse inhibition (PPI) of the acoustic startle reflex (ASR), locomotor activity and food preference [4].
• To elucidate the role of olfaction in this food preference, mice were subjected to an olfactory bulbectomy to remove the olfaction capability [5].

High impact information on Food Preferences

• Behavioral phenotyping indicated that GAL-tgs displayed normal general health and sensory and motor abilities; however, GAL-tg mice showed selective performance deficits on the Morris spatial navigational task and the social transmission of food preference olfactory memory test [6].
• Consistent with the idea that the Syt IV mutation preferentially affects hippocampal function, Syt IV mutant mice also display impaired social transmission of food preference [7].
• Circulating concentrations of magnesium, zinc, vitamin A, and alpha- and beta-carotene were measured, and food preferences were assessed by questionnaire [8].
• Remarkably, this effect of CREB function is not restricted to simple conditioning tasks, but also affects complex behaviours such as spatial memory and memory for socially transmitted food preferences [9].
• From 1030 until the meal at 1200 there was a significant satiating effect of intraduodenal fat compared with the control and loxiglumide experiments according to the food selection lists, which was because of the satiating effect for the fat rich items (p<0.05) [10].

Chemical compound and disease context of Food Preferences

• Children's food preferences and genetic sensitivity to the bitter taste of 6-n-propylthiouracil (PROP) [11].
• Enhanced choice of familiar food in a food preference test after chlordiazepoxide administration [12].
• Dietary GABA and food selection by rats [13].
• The data was incorporated into a control system model which depicts actions of spiperone in the food-preference situation with total eating duration as the final behavioural output [14].
• Chronic NE infusions into lesioned animals reversed the lesion-induced shift in relative food preference [15].

Biological context of Food Preferences

• In a double-blind, placebo-controlled study, we investigated the effect of the specific CCK-A receptor antagonist loxiglumide on food intake (carbohydrate-rich meal) and on subjective hunger feelings scored with visual analogue scales and food selection lists in seven healthy obese women and in seven healthy lean women [16].
• CREB phosphorylation and c-Fos expression in the hippocampus of rats during acquisition and recall of a socially transmitted food preference [17].
• We compared spontaneous caloric intake (SCI) of rats exclusively receiving chow to SCI of rats given the choice of chow or Parmesan cheese, and then examined the influence of parenteral feeding on SCI and food selection [18].
• Typology of food preferences identified by Nutri-Food Sort [19].
• The difference in food preferences resulted in a higher energy supply from carbohydrate and a lower intake of indigestible carbohydrate, vitamin B12, iron and iodine in patients than in controls [20].

Anatomical context of Food Preferences

• Role of noradrenaline and serotonin in the basolateral region of the amygdala in food preferences and learned taste aversions in the rat [21].

Gene context of Food Preferences

• Foxb1 mutant mice showed no deficits in such hippocampal-dependent tasks as contextual fear conditioning and social transmission of food preference [22].
• GAL transgenic (GAL-tg) mice overexpressing endogenous GAL show deficits on the probe trial of the Morris water maze spatial learning task, on the social transmission of food preference olfactory memory task, and on the trace cued fear conditioning emotional learning and memory task [23].
• Mice with a null mutation of the K+ channel regulatory subunit Kv beta 1.1 have altered excitability in hippocampal neurons and are impaired in the social transmission of food preferences (STFP) task [24].
• As it was possible that, unlike object recognition memory, novel odor recognition is not NMDA-receptor-dependent, a second task was designed using the social transmission of food preference paradigm [25].
• It is suggested that the cortical integration of olfactory and gustatory information could modulate mechanisms involved in food selection and emotional reactions relating to the chemical senses [26].

Analytical, diagnostic and therapeutic context of Food Preferences

• The effects of a tryptophan deficient amino acid mixture on food selection were studied using a double-blind counterbalanced crossover design in normal male subjects [27].

References