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MeSH Review

Dyskinesia, Drug-Induced

 
 
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Disease relevance of Dyskinesia, Drug-Induced

 

Psychiatry related information on Dyskinesia, Drug-Induced

 

High impact information on Dyskinesia, Drug-Induced

 

Chemical compound and disease context of Dyskinesia, Drug-Induced

 

Biological context of Dyskinesia, Drug-Induced

  • Analysis of covariance (ANCOVA), controlling for age at first antipsychotic treatment, revealed a significant effect of 5-HT2C genotype on orofacial dyskinesia (OFD) scores (F=3.47, df 2, P=.03) [18].
  • We investigated the role of oxidative stress in the pathophysiology of haloperidol (HP)-induced orofacial dyskinesia and evaluated the beneficial effect of Withania somnifera (Ws) root extract in the amelioration of HP-induced vacuous chewing movements (VCMs) and tongue protrusions in the rat model for TD [19].
 

Anatomical context of Dyskinesia, Drug-Induced

 

Gene context of Dyskinesia, Drug-Induced

  • Among patients withdrawn from neuroleptics, those with an oral dyskinesia had significantly lower peak GH concentration 2.46 +/- 0.93 ng/ml) after apomorphine compared with those without (14.85 +/- 3.83 ng/ml) (P less than 0.05) [24].
  • Effect of apomorphine on growth hormone and prolactin secretion in schizophrenic patients, with or without oral dyskinesia, withdrawn from chronic neuroleptic therapy [24].
  • Important role of striatal catalase in aging- and reserpine-induced oral dyskinesia [25].
  • OBJECTIVE: The objective of the present study was to elucidate the role of N-methyl-D-aspartate (NMDA) receptor involvement in neuroleptic-induced orofacial dyskinesia in rats [26].
  • The present results suggest that NK3 receptor-active peptides might be symptom inducers in oral dyskinesia [27].
 

Analytical, diagnostic and therapeutic context of Dyskinesia, Drug-Induced

References

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