Disease relevance of Lithiasis

• Biliary drug lithiasis: dipyridamole gallstones [1].
• BACKGROUND: Indinavir use is associated with a spectrum of renal and urinary tract complications including nephrolithiasis, renal colic and pain without recognizable lithiasis, and a picture of crystalluria-dysuria [2].
• Xanthine lithiasis, nephrocalcinosis, and renal failure in a leukemia patient treated with allopurinol [3].
• The medical treatment of nephrolithiasis is aimed in particular at the prevention of relapses, even though in some cases, such as cystine or uric acid lithiasis, the calculi also can be dissolved on site [4].
• CONCLUSION: It is argued that some patients with idiopathic pDBN have benign paroxysmal positional vertigo (BPPV) with lithiasis of the anterior canal [5].

High impact information on Lithiasis

• Triamterene lithiasis develops particularly in persons who have had a renal stone, and they should not receive this drug [6].
• We have reported previously two siblings with gout and uric acid lithiasis associated with excessive purine production [7].
• Does total parenteral nutrition induce gallbladder sludge formation and lithiasis [8]?
• Here we describe the first case of DHA lithiasis in a patient who has functional APRT activity in cultured lymphoblasts [9].
• Female Sprague-Dawley rats weighing 45-55 g fed a purified diet for 18 days developed hydroxyapatite intratubular lithiasis, the earliest calcific lesions being detectable by light microscopy on Day 12 [10].

Chemical compound and disease context of Lithiasis

• 2,8-Dihydroxyadenine lithiasis in a Japanese patient heterozygous at the adenine phosphoribosyltransferase locus [9].
• Biliary lithiasis appeared in two patients after six months of lanreotide [11].
• Lopinavir-ritonavir (Kaletra) and lithiasis: seven cases [12].
• Even rare components such as cystine and xanthine were unraveled in London. The impetus given by these studies offered a good start to clinical chemistry in general and to the understanding of urinary lithiasis in particular [13].
• Ceftriaxone may cause reversible biliary pseudolithiasis, notably at higher dosages of the drug (>/=2 g/day); however, the incidence of true lithiasis is <0.1% [14].

Biological context of Lithiasis

• The 24-hour urinary excretion of various electrolytes, as well as the glomerular filtration rate, renal plasma flow and maximal rate of tubular excretion of para-aminohippuric acid was measured in each kidney of 10 patients with idiopathic renal lithiasis [15].
• Case from the aerospace medicine residents' teaching file. Case #35. Renal cell carcinoma in an aviator with a long history of renal lithiasis [16].

Anatomical context of Lithiasis

• In the present investigations, we devised a rapid method using erythrocytes for the diagnosis of partial APRT deficiencies accompanied by severe impairment in adenine metabolism causing 2,8-dihydroxyadenine lithiasis [17].
• Effect of short-term octreotide therapy and total parenteral nutrition on the development of biliary sludge and lithiasis [18].
• The most severe developments were observed in common bile duct lithiasis or in patients with tumors who underwent PTC [19].

Gene context of Lithiasis

• Mice with adenine phosphoribosyltransferase deficiency develop fatal 2,8-dihydroxyadenine lithiasis [20].
• During fasting, plasma pancreatic polypeptide concentrations were significantly higher in lithiasis patients (P < 0.03) [21].
• Renal lithiasis and hyperparathyroidism: diagnosis, management and prognosis [22].
• Partially purified APRT from one of the Japanese families with the lithiasis associated with a partial deficiency of APRT had a reduced affinity for 5-phosphoribosyl-1-pyrophosphate (PRPP) [23].
• We recommend trying PFPP when the side of lithiasis cannot be determined, in cases that are resistant to particle repositioning canal manoeuvres, and before considering canal plugging for refractory antBPPV [24].

Analytical, diagnostic and therapeutic context of Lithiasis

• From February 1983 through 1986, 15 patients (17 renal units) with cystine urinary lithiasis were treated by percutaneous ultrasonic lithotripsy [25].
• Double-blind study with thiazide in recurrent calcium lithiasis [26].
• The purpose of the present study was to investigate the incidence of biliary lithiasis 6 and 12 months after treatment with ceftriaxone and to compare it with that in patients treated with amoxycillin/clavulanate [27].
• Sulfadiazine-related obstructive urinary tract lithiasis: an unusual cause of acute renal failure after kidney transplantation [28].
• 5. CONCLUSION: From the obtained results it can be concluded that the test is useful to evaluate the efficacy of a given renal lithiasis medical treatment, and also the efficacy of the treatment of calcium oxalate renal lithiasis using crystallization inhibitors, such as citrate and phytate, in an important number of cases [29].

References