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MeSH Review

Renal Plasma Flow

 
 
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Disease relevance of Renal Plasma Flow

 

Psychiatry related information on Renal Plasma Flow

 

High impact information on Renal Plasma Flow

 

Chemical compound and disease context of Renal Plasma Flow

 

Biological context of Renal Plasma Flow

 

Anatomical context of Renal Plasma Flow

 

Associations of Renal Plasma Flow with chemical compounds

 

Gene context of Renal Plasma Flow

  • Although it is unlikely that PTHrP regulates systemic hemodynamics, its chronotropic effect and its potent action on renal plasma flow may represent the primary cardiovascular physiological targets of action [29].
  • Infusion of PYY at the higher dose, which reproduced plasma levels seen during a diarrheal illness, resulted in similar changes in renal function and also reduced renal plasma flow by approximately 10% (P < 0.05) [30].
  • Infusion of PYY into rats (47 pmol x kg(-1) x min[-1]) increased mean arterial pressure (103 +/- 6 to 123 +/- 8 mmHg) and decreased renal plasma flow (13 +/- 1.8 to 8.4 +/- 2.1 ml/min) but produced no significant change in glomerular filtration rate or sodium excretion [31].
  • Despite the lower MAP, the glomerular filtration rate (GFR), renal plasma flow (RPF), absolute sodium excretion, and fractional sodium excretion were not significantly different between wild-type and AT1A-/- mice [32].
  • Glomerular filtration rate, renal plasma flow, and albumin excretion rate were measured together with plasma concentrations of glucagon, growth hormone, insulin-like growth factor 1 (IGF-1), somatostatin, serum insulin and free amino acids in blood [33].
 

Analytical, diagnostic and therapeutic context of Renal Plasma Flow

References

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