Disease relevance of Reaction product

• Reaction product within the inner plexiform layer was observed in processes of ganglion cells at their sites of input from cone bipolar cells [1].

High impact information on Reaction product

• Reaction product was visualized as very fine particulate within the cisternae of the nuclear envelope and endoplasmic reticulum [2].
• Reaction product was intense surrounding most villus capillaries but, when present, was faint in dome capillary adventitia [3].
• Reaction product was consistenly found in dendrites and in association with the cytoplasmic aspect of the dendritic plasmalemma [4].
• Reaction product often filled portions of the tubular smooth endoplasmic reticulum in these processes [4].
• Reaction product, which forms when cerous ions react with H2O2 to form an electron-dense precipitate, was demonstrated on the cell surface and within the phagosomes of phagocytically stimulated cells when D-amino acids were provided as substrate [5].

Biological context of Reaction product

• Reaction product intensity and cell size were measured [6].
• Reaction product from hydrolysis of 3'-NADP+, however, was localized at a different site, that is, within granules and membranous connections of the GERL system in the cell [7].
• Reaction product formation was much more complex than nitrosation of the parent molecule dimethylamino moiety to form nitrosodimethylamine [8].
• Reaction product of the enzyme activity was found confined to the extracellular surface of the plasma membrane of cardiac myocytes and endothelial cells due to the corresponding orientation of the enzyme active sites [9].
• These RP-labelled organelles were only localized in the supra- or para-nuclear regions of the ameloblasts, which corresponds with previous studies using acid phosphatase cytochemistry [10].

Anatomical context of Reaction product

• Reaction product was also present in secondary lysosomes [11].
• Reaction product was confined to synaptic vesicles, as predicted [12].
• Reaction product was noted along membranes of vascular endothelial cells and interstitial fibroblasts, the sites of early calcific deposits in bioprosthetic valves, by ultrastructural examination of GLUT-treated tissue [13].
• Reaction product diffusely labeled the cytoplasm of glial cell bodies and processes after intraneuronal injection of HRP [14].
• Reaction product for ICAM-1 was observed associated with some but not all blood vessels of the tumors examined [15].

Gene context of Reaction product

• Reaction product after anti-PLP-peptide was restricted to infrequent bizarre cells possibly representing abnormal oligodendroglia [16].
• Reaction product, though sparse, was found in the somata of Purkinje cells, primarily in the region of the Golgi apparatus [17].
• Reaction product indicative of the presence of somatostatin in presumptive nerve endings was located on the ventral surface of the median eminence and in the external lamina of the infundibulum in proximity to the superficial portal capillaries [18].
• Reaction product deposition attributable to catalase, glycolate oxidase, and urate oxidase activities are cytochemically localized to Yucca root microbodies and classifies them as unspecialized peroxisomes on the basis of their enzyme complement and tissue origin [19].
• Reaction product was also identified in granulocyte-macrophage clones, presumably reflecting the content of other heme proteins such as catalase and cytochromes [20].

Analytical, diagnostic and therapeutic context of Reaction product

• Reaction product analysis by HPLC clearly indicated that BGN16.1 has an endo-hydrolytic mode of action [21].
• Reaction product identification and quantification are accomplished simultaneously with an enzyme-linked immunosorbent assay (ELISA) using carbohydrate sequence-specific monoclonal antibodies. beta-Galactosyltransferase and alpha-galactosidase reactions were used to illustrate the salient features of the method [22].
• Electron microscopy revealed that at E13 peroxidase reaction product for GD3 (RP) was present on the plasma membrane and in the cytoplasm of neuroblasts, with accentuation in the former [23].
• Reaction product of 5-OHDA was observed along the endolymphatic surface of the cochlear duct in both 10-minute and 30-minute perfusions, while that of HRP was observed only in the 30-minute perfusion [24].
• Reaction product for TG coincided well with previous investigations studied by electron microscopic autoradiography and is strongly positive in most follicular lumina having no relation to their size, reabsorbed colloid droplets or small subapical vesicles [25].

References