Disease relevance of Crassostrea

• Twenty bacteriophages active against Vibrio parahaemolyticus and agar-digesting vibrios, isolated from oysters (Crassostrea gigas) and Dungeness crab (Cancer magister) and by induction of a lysogenic agar digester, were tested as to their host range [1].
• Promoters from Drosophila heat shock protein and cytomegalovirus drive transient expression of luciferase introduced by particle bombardment into embryos of the oyster Crassostrea gigas [2].
• Aspects of mitochondrial activity in the estuarine bivalves Crassostrea rhizophorae and Lucina pectinatus: a comparative approach [3].
• Phototoxicity of pyrene and benzo[a]pyrene to embryo-larval stages of the Pacific oyster Crassostrea gigas [4].

High impact information on Crassostrea

• In response to the neurohormone serotonin (5-hydroxytryptamine, 5-HT), prophase-arrested oocytes of the marine bivalve Crassostrea gigas (oyster) reinitiate meiosis, undergo germinal vesicle breakdown (GVBD), and are arrested again at metaphase I [5].
• We report here the identification and functional characterization of Cg-Rel, a gene encoding the Crassostrea gigas homolog of Rel/NF-kappaB transcription factors found in insects and mammals [6].
• Study of atrazine effects on Pacific oyster, Crassostrea gigas, haemocytes [7].
• cDNA cloning and molecular identification of the major oyster allergen from the Pacific oyster Crassostrea gigas [8].
• Cholinesterases from the common oyster (Crassostrea gigas). Evidence for the presence of a soluble acetylcholinesterase insensitive to organophosphate and carbamate inhibitors [9].

Chemical compound and disease context of Crassostrea

• Use of diacetyl to reduce the load of Vibrio vulnificus in the Eastern oyster, Crassostrea virginica [10].

Biological context of Crassostrea

• Effects of acclimation temperature and cadmium exposure on cellular energy budgets in the marine mollusk Crassostrea virginica: linking cellular and mitochondrial responses [11].
• We observed in vitro that after treatment with the Arg-Gly-Asp (RGD) peptide, non-spreading Crassostrea gigas hemocytes underwent cell death [12].
• Comparison with heat shock responses in tissues of Crassostrea gigas indicated a similar pattern of Hsp70 expression [13].
• Following repeated attempts to resolve these differences using the Pacific oyster (Crassostrea gigas) embryo development assay, a 24-h EC50 of 170 mg BrO3- liter-1 was observed [14].
• Eastern oysters (Crassostrea virginica) were exposed to [14C]chlorpyrifos (O,O-diethyl-O-[3,5,6-trichloro-2-pyridyl] phosphorothioate) at an average measured seawater concentration of 0.6 microg/L under flow-through conditions for 28 d [15].

Anatomical context of Crassostrea

• Noradrenaline modulates hemocyte reactive oxygen species production via beta-adrenergic receptors in the oyster Crassostrea gigas [16].
• Cadmium distribution was studied in different subcellular fractions of gill and hepatopancreas tissues of eastern oysters Crassostrea virginica [17].
• A study of steady state and kinetic regulation of chloride ion and osmotic pressure in hemolymph of oysters, Crassostrea virginica, exposed to tri-n-butyltin [18].

Associations of Crassostrea with chemical compounds

• Purification and characterization studies of cadmium-binding proteins from the American oyster, Crassostrea virginica [19].
• The negative chronotropic effects of ACh, but not of 4-ketoamyltrimethylammonium, were potentiated by DMSO (1-5 microliters/ml) on the isolated heart of the oyster Crassostrea virginica [20].
• Molecular data support a later divergence of the tropical Pacific Saccostrea from a common ancestor of the Atlantic Crassostrea species [21].
• Techniques for delivery of arachidonic acid to Pacific oyster, Crassostrea gigas, spat [22].
• Alanopine and strombine are novel imino acids produced by a dehydrogenase found in the adductor muscle of the oyster, Crassostrea gigas [23].

Gene context of Crassostrea

• Molecular characterization of a cDNA encoding putative vitellogenin from the Pacific oyster Crassostrea gigas [24].
• Structure and expression of mGDF, a new member of the transforming growth factor-beta superfamily in the bivalve mollusc Crassostrea gigas [25].
• 1. Influence on the Biochemical and Fatty Acid Compositions of Raw Oysters (Crassostrea gigas) [26].
• Two BHC bioconcentration studies were conducted with the oyster, Crassostrea virginica, and pinfish [27].
• Insulin-like system and growth regulation in the Pacific oyster Crassostrea gigas: hrIGF-1 effect on protein synthesis of mantle edge cells and expression of an homologous insulin receptor-related receptor [28].

Analytical, diagnostic and therapeutic context of Crassostrea

• Two low-molecular-mass cadmium-induced, cadmium-, zinc-binding proteins were purified from the oyster Crassostrea virginica using procedures that included acetone precipitation, Sephadex gel chromatography, and anion-exchange and reverse-phase high-performance liquid chromatography [29].
• However, the results of competitive ELISA inhibition experiments suggest that Tur c 1 has an IgE-binding epitope in the C-terminal region which is dissimilar to those proposed for Cra g 1 (the oyster Crassostrea gigas allergen) and Pen i 1 (the shrimp Penaeus indicus allergen) [30].

References