Disease relevance of Fumigation

• This incident stresses the need for improved worker education and strict adherence to safety precautions during all stages of methyl bromide fumigation and raises the possibility of an increased risk of toxicity associated with methyl bromide fumigation during a cool season [1].
• The effect of relative humidity on swine vesicular disease virus in dried films before and during formaldehyde fumigation [2].
• Body weight, mortality and behaviour (eating, drinking, sitting and standing activities) were not affected by formaldehyde fumigation [3].
• Observations on the effects of formaldehyde on cockroaches and their flora: I. Survival of vaccinia virus-infected cockroaches during fumigation with formaldehyde [4].

High impact information on Fumigation

• After 3 years of CO2 fumigation, trees were twice as likely to be reproductively mature and produced three times as many cones and seeds as trees at ambient CO2 concentration [5].
• Consistent with the proposed role for ascorbate in reactive oxygen species detoxification, lipid peroxides are elevated in soz1, but not in wild type following ozone fumigation [6].
• Agricultural soil fumigation as a source of atmospheric methyl bromide [7].
• These data further suggest that fumigation with the relatively low atmospheric concentrations of monoterpenes that are occasionally observed during warm windless days in the Mediterranean canopies may significantly improve the heat tolerance of nonemitting vegetation that grows intermixed with emitting species [8].
• Differential effects of elevated ozone on two hybrid aspen genotypes predisposed to chronic ozone fumigation. Role of ethylene and salicylic acid [9].

Biological context of Fumigation

• OBJECTIVES: This study assessed the health effects associated with occupational exposure to methyl bromide and sulfuryl fluoride among structural fumigation workers [10].
• Terminal disinfection of calf houses by formaldehyde fumigation [11].
• Use of methyl bromide for pest control fumigation may result in adverse exposure to three populations: the actual fumigators; other workers not actually involved in the fumigation; and the general public in the vicinity [12].
• Air pollution and agricultural aphid pests. I: Fumigation experiments with SO2 and NO2 [13].
• Infection control measures included ward closure followed by thorough cleaning, and formaldehyde fumigation [14].

Anatomical context of Fumigation

• In BTH-pretreated, but not fumigated, plants, H(2)O(2) accumulation occurred in the cell walls and no dead cells were detected, whereas O(3) fumigation of untreated plants produced H(2)O(2) accumulation also inside some palisade mesophyll cells, causing their death [15].
• The fumigation programme was started with methyl bromide treatment directed primarily against arthropods, followed by ammonia spray to kill coccidial oocysts and concluded by three formaldehyde treatments with fog and spray against bacteria and viruses [16].

Associations of Fumigation with chemical compounds

• Upon fumigation with 0.1 microL L(-1) sulfur dioxide, the Cys and glutathione contents in leaves of F(1) plants were increased significantly, but not in leaves of non-transformed control plants [17].
• The second of the two [Ca2+]cyt peaks, but not the first, could be eliminated either by pre-treatment of plants with lanthanum chloride, or by reducing the duration of ozone fumigation [18].
• The isoprene protection effect was directly demonstrated by fumigation of young (non-emitting) leaves, treated with RB or bromoxynil (simulating photoinhibition) [19].
• Fumigation experiments with toxic levels of hydrogen sulphide (H2S) gas showed that while sense transformants were highly resistant, control and antisense plants were severely damaged by the treatment [20].
• Total phenolic content was also strongly stimulated at the 10th and 72nd h from starting fumigation, concomitant with an enhancement in phenylalanine ammonia-lyase a and phenylalanine ammonia-lyase b expression, as evaluated by reverse transcriptase-polymerase chain reaction [21].

Gene context of Fumigation

• Incubation with 1-aminocyclopropane-1-carboxylic acid, the precursor of ethylene, increased while fumigation with ethylene decreased expression and activity of ST and CAS [22].
• Ascorbate peroxidase activity increased 1.8-fold without a lag period during fumigation with 0.1 ppm ozone, while guaiacol peroxidase activity increased 4.4-fold with a 1-day lag [23].
• In addition, fumigation workers had significantly reduced performance on the Santa Ana Dexterity Test of the dominant hand and a nonsignificantly higher prevalence of carpal tunnel syndrome than did the referents [10].
• Plastic tarps currently used during soil fumigation to control emissions have been shown to be permeable to fumigant vapors, resulting in appreciable losses to the atmosphere [24].
• The concentration of ascorbate (ASC) was determined in leaf extracellular washing fluid (apoplast) and in the residual leaf tissue (symplast) after 0, 4 and 8 h acute fumigation, and after a 16 h "recovery" period in CFA [25].

Analytical, diagnostic and therapeutic context of Fumigation

• Bioreactors for removing methyl bromide following contained fumigations [26].
• Precocene II induced precocious metamorphosis in both species when applied by either contact exposure or fumigation, and this effect could be prevented by juvenile hormone replacement therapy [27].
• The acaricidal activities of materials derived from the root bark of Paeonia suffruticosa against adults of Dermatophagoides farinae and Dermatophagoides pteronyssinus were examined using direct contact and fumigation bioassays and compared with those of benzyl benzoate, dibutyl phthalate, and N,N-diethyl-m-toluamide (deet), widely used acaricides [28].
• This paper describes the application of these methods using the example of our recent cross-sectional study of workers occupationally exposed to ethylene dibromide in the papaya fumigation industry [29].

References