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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 
MeSH Review

Syncope, Vasovagal

 
 
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Disease relevance of Syncope, Vasovagal

 

High impact information on Syncope, Vasovagal

 

Chemical compound and disease context of Syncope, Vasovagal

 

Biological context of Syncope, Vasovagal

 

Anatomical context of Syncope, Vasovagal

  • Because these terminals mediate vasovagal syncope, these data could suggest a mechanistic role of extracellular ATP in this syndrome and, in addition, give further support to the hypothesis that endogenous ATP released from ischemic myocytes is a mediator of atropine-sensitive bradyarrhythmias associated with left ventricular myocardial infarction [16].
  • In conclusion, the efficacy of beta-blockers is associated not only with a reduction of the sympathoadrenal stimulation seen in vasovagal syncope but also with a lower release of VP suggesting that low-pressure baroreceptors might be involved in VP release [17].
  • The increase in the levels of E and HVA both at baseline and after the tilt test, without a corresponding rise in NE levels indicates enhanced activity of the adrenomedullary axis which is not paralleled by NE release from sympathetic nerve endings in patients of neurocardiogenic syncope [18].
 

Gene context of Syncope, Vasovagal

 

Analytical, diagnostic and therapeutic context of Syncope, Vasovagal

References

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