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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 
MeSH Review

Sleep Deprivation

 
 
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Disease relevance of Sleep Deprivation

 

Psychiatry related information on Sleep Deprivation

 

High impact information on Sleep Deprivation

  • Consistent with the protective effect of heat-shock genes, was the observation that flies carrying a mutation for the heat-shock protein Hsp83 (Hsp83(08445)) showed exaggerated homeostatic response and died after sleep deprivation [11].
  • During sleep deprivation, glucose levels and insulin secretion rose again to reach a maximum at a time corresponding to the beginning of the habitual sleep period [12].
  • After sleep deprivation, the blunting of the normal sleep-related growth-hormone (GH) pulse is compensated during the day [13].
  • We measured serum thyrotropin every 15 minutes during the night of sleep deprivation, thyrotropin bioactivity, the thyrotropin response to protirelin the next afternoon, and other indexes of hypothalamic-pituitary-thyroid function [14].
  • Effects of tryptophan depletion in drug-free depressed patients who responded to total sleep deprivation [15].
 

Chemical compound and disease context of Sleep Deprivation

 

Biological context of Sleep Deprivation

 

Anatomical context of Sleep Deprivation

 

Gene context of Sleep Deprivation

  • This study compares cortical EEG and architecture of the sleep-waking cycle in adult Ube3a m-/p+ mice compared with those of age-matched WT (m+/p+) mice, under baseline conditions or after 4-h sleep deprivation (SD) [30].
  • Therefore, we studied the effect of sleep deprivation on nocturnal ghrelin secretion [31].
  • Sleep deprivation delayed the normal night time increase in gastric TFF2 and resulted in an overall reduction in TFF2 secretion [32].
  • Inhibition of either IL-1 or TNF inhibits spontaneous sleep and the sleep rebound that occurs after sleep deprivation [33].
  • The studies include profiles of GH secretion in normal men and women in fed and fasted states; analysis of GH secretion during sleep; and administration of GHRH during different stages of sleep and after sleep deprivation [34].
 

Analytical, diagnostic and therapeutic context of Sleep Deprivation

  • We investigated its role in sleep and sleep regulation by comparing baseline recordings and the effects of sleep deprivation in PrP knockout mice (129/SV) and wild-type controls (129/SV x C57BL/6), which are the mice used for most gene targeting experiments and whose behavior is not well characterized [35].
  • METHOD: Regional cerebral glucose metabolism was assessed by positron emission tomography (PET) with [18F]deoxyglucose (FDG) before and after total sleep deprivation in 15 unmedicated awake patients with unipolar major depression and 15 normal control subjects, who did the continuous performance test during FDG uptake [36].
  • The changes in non-REM sleep EEG spectra are dissimilar from the spectral changes induced by sleep deprivation and exhibit a close temporal association with the melatonin rhythm and the endogenous circadian phase of sleep consolidation [37].
  • The interventions studied included antidepressant medication, estrogen therapy, critically timed sleep deprivation, and bright light therapy [38].
  • Response to total sleep deprivation before and during treatment with fluvoxamine or maprotiline in patients with major depression--results of a double-blind study [39].

References

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