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Encephalomyelitis, Autoimmune, Experimental

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  1. A critical role for the protein tyrosine phosphatase receptor type Z in functional recovery from demyelinating lesions. Harroch, S., Furtado, G.C., Brueck, W., Rosenbluth, J., Lafaille, J., Chao, M., Buxbaum, J.D., Schlessinger, J. Nat. Genet. (2002) [Pubmed]
  2. CNTF is a major protective factor in demyelinating CNS disease: a neurotrophic cytokine as modulator in neuroinflammation. Linker, R.A., Mäurer, M., Gaupp, S., Martini, R., Holtmann, B., Giess, R., Rieckmann, P., Lassmann, H., Toyka, K.V., Sendtner, M., Gold, R. Nat. Med. (2002) [Pubmed]
  3. Differential tumor necrosis factor alpha expression by astrocytes from experimental allergic encephalomyelitis-susceptible and -resistant rat strains. Chung, I.Y., Norris, J.G., Benveniste, E.N. J. Exp. Med. (1991) [Pubmed]
  4. The complement inhibitory protein DAF (CD55) suppresses T cell immunity in vivo. Liu, J., Miwa, T., Hilliard, B., Chen, Y., Lambris, J.D., Wells, A.D., Song, W.C. J. Exp. Med. (2005) [Pubmed]
  5. A peptide-binding motif for I-A(g7), the class II major histocompatibility complex (MHC) molecule of NOD and Biozzi AB/H mice. Harrison, L.C., Honeyman, M.C., Trembleau, S., Gregori, S., Gallazzi, F., Augstein, P., Brusic, V., Hammer, J., Adorini, L. J. Exp. Med. (1997) [Pubmed]
  6. Central serotonin receptor sensitivity in rats with experimental allergic encephalomyelitis. White, S.R., Bieger, D. Res. Commun. Chem. Pathol. Pharmacol. (1980) [Pubmed]
  7. Identification of murine loci associated with susceptibility to chronic experimental autoimmune encephalomyelitis. Sundvall, M., Jirholt, J., Yang, H.T., Jansson, L., Engström, A., Pettersson, U., Holmdahl, R. Nat. Genet. (1995) [Pubmed]
  8. Lipid microarrays identify key mediators of autoimmune brain inflammation. Kanter, J.L., Narayana, S., Ho, P.P., Catz, I., Warren, K.G., Sobel, R.A., Steinman, L., Robinson, W.H. Nat. Med. (2006) [Pubmed]
  9. Suppression of experimental autoimmune encephalomyelitis by selective blockade of encephalitogenic T-cell infiltration of the central nervous system. Yan, S.S., Wu, Z.Y., Zhang, H.P., Furtado, G., Chen, X., Yan, S.F., Schmidt, A.M., Brown, C., Stern, A., LaFaille, J., Chess, L., Stern, D.M., Jiang, H. Nat. Med. (2003) [Pubmed]
  10. Class IV semaphorin Sema4A enhances T-cell activation and interacts with Tim-2. Kumanogoh, A., Marukawa, S., Suzuki, K., Takegahara, N., Watanabe, C., Ch'ng, E., Ishida, I., Fujimura, H., Sakoda, S., Yoshida, K., Kikutani, H. Nature (2002) [Pubmed]
  11. Cbl-b regulates the CD28 dependence of T-cell activation. Chiang, Y.J., Kole, H.K., Brown, K., Naramura, M., Fukuhara, S., Hu, R.J., Jang, I.K., Gutkind, J.S., Shevach, E., Gu, H. Nature (2000) [Pubmed]
  12. Prevention of experimental autoimmune encephalomyelitis by antibodies against alpha 4 beta 1 integrin. Yednock, T.A., Cannon, C., Fritz, L.C., Sanchez-Madrid, F., Steinman, L., Karin, N. Nature (1992) [Pubmed]
  13. TNF is a potent anti-inflammatory cytokine in autoimmune-mediated demyelination. Liu, J., Marino, M.W., Wong, G., Grail, D., Dunn, A., Bettadapura, J., Slavin, A.J., Old, L., Bernard, C.C. Nat. Med. (1998) [Pubmed]
  14. Oral tolerance to myelin basic protein and natural recovery from experimental autoimmune encephalomyelitis are associated with downregulation of inflammatory cytokines and differential upregulation of transforming growth factor beta, interleukin 4, and prostaglandin E expression in the brain. Khoury, S.J., Hancock, W.W., Weiner, H.L. J. Exp. Med. (1992) [Pubmed]
  15. Isoprenoids determine Th1/Th2 fate in pathogenic T cells, providing a mechanism of modulation of autoimmunity by atorvastatin. Dunn, S.E., Youssef, S., Goldstein, M.J., Prod'homme, T., Weber, M.S., Zamvil, S.S., Steinman, L. J. Exp. Med. (2006) [Pubmed]
  16. Inhibition of leukotriene B4-receptor interaction suppresses eosinophil infiltration and disease pathology in a murine model of experimental allergic encephalomyelitis. Gladue, R.P., Carroll, L.A., Milici, A.J., Scampoli, D.N., Stukenbrok, H.A., Pettipher, E.R., Salter, E.D., Contillo, L., Showell, H.J. J. Exp. Med. (1996) [Pubmed]
  17. BTLA is a lymphocyte inhibitory receptor with similarities to CTLA-4 and PD-1. Watanabe, N., Gavrieli, M., Sedy, J.R., Yang, J., Fallarino, F., Loftin, S.K., Hurchla, M.A., Zimmerman, N., Sim, J., Zang, X., Murphy, T.L., Russell, J.H., Allison, J.P., Murphy, K.M. Nat. Immunol. (2003) [Pubmed]
  18. Challenging cytokine redundancy: inflammatory cell movement and clinical course of experimental autoimmune encephalomyelitis are normal in lymphotoxin-deficient, but not tumor necrosis factor-deficient, mice. Sean Riminton, D., Körner, H., Strickland, D.H., Lemckert, F.A., Pollard, J.D., Sedgwick, J.D. J. Exp. Med. (1998) [Pubmed]
  19. Leptin surge precedes onset of autoimmune encephalomyelitis and correlates with development of pathogenic T cell responses. Sanna, V., Di Giacomo, A., La Cava, A., Lechler, R.I., Fontana, S., Zappacosta, S., Matarese, G. J. Clin. Invest. (2003) [Pubmed]
  20. Regulation of experimental autoimmune encephalomyelitis with insulin-like growth factor (IGF-1) and IGF-1/IGF-binding protein-3 complex (IGF-1/IGFBP3). Lovett-Racke, A.E., Bittner, P., Cross, A.H., Carlino, J.A., Racke, M.K. J. Clin. Invest. (1998) [Pubmed]
  21. Insulin-like growth factor I treatment reduces demyelination and up-regulates gene expression of myelin-related proteins in experimental autoimmune encephalomyelitis. Yao, D.L., Liu, X., Hudson, L.D., Webster, H.D. Proc. Natl. Acad. Sci. U.S.A. (1995) [Pubmed]
  22. Selection for T-cell receptor V beta-D beta-J beta gene rearrangements with specificity for a myelin basic protein peptide in brain lesions of multiple sclerosis. Oksenberg, J.R., Panzara, M.A., Begovich, A.B., Mitchell, D., Erlich, H.A., Murray, R.S., Shimonkevitz, R., Sherritt, M., Rothbard, J., Bernard, C.C. Nature (1993) [Pubmed]
  23. Transfer of experimental allergic encephalomyelitis with guinea pig peritoneal exudate cells. Driscoll, B.F., Kies, M.W., Alvord, E.C. Science (1979) [Pubmed]
  24. Human nerve growth factor protects common marmosets against autoimmune encephalomyelitis by switching the balance of T helper cell type 1 and 2 cytokines within the central nervous system. Villoslada, P., Hauser, S.L., Bartke, I., Unger, J., Heald, N., Rosenberg, D., Cheung, S.W., Mobley, W.C., Fisher, S., Genain, C.P. J. Exp. Med. (2000) [Pubmed]
  25. Antigen-driven bystander suppression after oral administration of antigens. Miller, A., Lider, O., Weiner, H.L. J. Exp. Med. (1991) [Pubmed]
  26. Absence of monocyte chemoattractant protein 1 in mice leads to decreased local macrophage recruitment and antigen-specific T helper cell type 1 immune response in experimental autoimmune encephalomyelitis. Huang, D.R., Wang, J., Kivisakk, P., Rollins, B.J., Ransohoff, R.M. J. Exp. Med. (2001) [Pubmed]
  27. Requirement for CD40 ligand in costimulation induction, T cell activation, and experimental allergic encephalomyelitis. Grewal, I.S., Foellmer, H.G., Grewal, K.D., Xu, J., Hardardottir, F., Baron, J.L., Janeway, C.A., Flavell, R.A. Science (1996) [Pubmed]
  28. Lymphoid/neuronal cell surface OX2 glycoprotein recognizes a novel receptor on macrophages implicated in the control of their function. Wright, G.J., Puklavec, M.J., Willis, A.C., Hoek, R.M., Sedgwick, J.D., Brown, M.H., Barclay, A.N. Immunity (2000) [Pubmed]
  29. Studies in B7-deficient mice reveal a critical role for B7 costimulation in both induction and effector phases of experimental autoimmune encephalomyelitis. Chang, T.T., Jabs, C., Sobel, R.A., Kuchroo, V.K., Sharpe, A.H. J. Exp. Med. (1999) [Pubmed]
  30. Dual role for Fas ligand in the initiation of and recovery from experimental allergic encephalomyelitis. Sabelko-Downes, K.A., Cross, A.H., Russell, J.H. J. Exp. Med. (1999) [Pubmed]
  31. A critical role for lymphotoxin in experimental allergic encephalomyelitis. Suen, W.E., Bergman, C.M., Hjelmström, P., Ruddle, N.H. J. Exp. Med. (1997) [Pubmed]
  32. Peripheral T cells are the therapeutic targets of glucocorticoids in experimental autoimmune encephalomyelitis. Wüst, S., van den Brandt, J., Tischner, D., Kleiman, A., Tuckermann, J.P., Gold, R., Lühder, F., Reichardt, H.M. J. Immunol. (2008) [Pubmed]
  33. Vaccination with DNA encoding an immunodominant myelin basic protein peptide targeted to Fc of immunoglobulin G suppresses experimental autoimmune encephalomyelitis. Lobell, A., Weissert, R., Storch, M.K., Svanholm, C., de Graaf, K.L., Lassmann, H., Andersson, R., Olsson, T., Wigzell, H. J. Exp. Med. (1998) [Pubmed]
  34. Antigen-specific therapy of experimental allergic encephalomyelitis by soluble class II major histocompatibility complex-peptide complexes. Sharma, S.D., Nag, B., Su, X.M., Green, D., Spack, E., Clark, B.R., Sriram, S. Proc. Natl. Acad. Sci. U.S.A. (1991) [Pubmed]
  35. Pathogenic myelin oligodendrocyte glycoprotein antibodies recognize glycosylated epitopes and perturb oligodendrocyte physiology. Marta, C.B., Oliver, A.R., Sweet, R.A., Pfeiffer, S.E., Ruddle, N.H. Proc. Natl. Acad. Sci. U.S.A. (2005) [Pubmed]
  36. Antineuroinflammatory effect of NF-kappaB essential modifier-binding domain peptides in the adoptive transfer model of experimental allergic encephalomyelitis. Dasgupta, S., Jana, M., Zhou, Y., Fung, Y.K., Ghosh, S., Pahan, K. J. Immunol. (2004) [Pubmed]
  37. Gene therapy for chronic relapsing experimental allergic encephalomyelitis using cells expressing a novel soluble p75 dimeric TNF receptor. Croxford, J.L., Triantaphyllopoulos, K.A., Neve, R.M., Feldmann, M., Chernajovsky, Y., Baker, D. J. Immunol. (2000) [Pubmed]
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