The world's first wiki where authorship really matters (Nature Genetics, 2008). Due credit and reputation for authors. Imagine a global collaborative knowledge base for original thoughts. Search thousands of articles and collaborate with scientists around the globe.

wikigene or wiki gene protein drug chemical gene disease author authorship tracking collaborative publishing evolutionary knowledge reputation system wiki2.0 global collaboration genes proteins drugs chemicals diseases compound
Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
MeSH Review

Burn Units

Welcome! If you are familiar with the subject of this article, you can contribute to this open access knowledge base by deleting incorrect information, restructuring or completely rewriting any text. Read more.

Disease relevance of Burn Units


High impact information on Burn Units

  • RESULTS--A clonal relationship demonstrated among isolates from burn unit patients and staff was clearly distinguishable from MRSA isolates arising from other hospital wards [6].
  • After a focal outbreak of methicillin-resistant S aureus in the burn unit, acquisition of the organism by patients on other wards increased significantly [7].
  • Seventy-nine patients admitted to a major regional burn center were randomly assigned to groups that received 1 mg of lorazepam or a placebo in addition to their standard opioid analgesics [8].
  • Emergence in a burn center of populations of bacteria resistant to gentamicin, tobramycin, and amikacin: evidence for the need for changes in zone diameter interpretative standards [9].
  • Taking a cut-off value of 110 unit burn standard for severity of injury, plasma phenylalanine concentrations, and urinary 3-methylhistidine/creatinine ratio were significantly reduced (p < .05) in the less severe burn patients (<110 unit burn standard) supplemented with ornithine alpha-ketoglutarate [10].

Chemical compound and disease context of Burn Units


Biological context of Burn Units


Associations of Burn Units with chemical compounds

  • The second group (n = 15) was treated conservatively with silver sulfadiazine in other burn facilities for 5 days, and burn wounds were surgically excised when patients were admitted to our burn center on day 6 after the injury [17].
  • When examined by individual ICU, however, imipenem resistance was seen in the general medicine and burn units [18].
  • At this burn center, sulfonamide resistance occurred in more than 80% of gram-negative isolates [19].
  • CONCLUSIONS: These data indicate that the sulfonamides and phenytoin are the most common etiologic agents, expeditious transfer to a burn center reduces morbidity, and corticosteroid administration dramatically increases mortality [20].
  • PURPOSE: To evaluate the relative contribution of methamphetamine production-related accidents to facial and ocular injuries admitted to a regional burn unit [21].

Gene context of Burn Units

  • This study indicates a high incidence of staphylococcal septicaemia (especially due to MRSA) in the burn unit [22].
  • ProCT and IL6 are prognostic factors of mortality at the time of admission but less reliable than the clinical UBS (unit burn standard) score [23].
  • In the burn unit, MARS were recovered from the air, from the hair and hands of personnel, and from inanimate objects [24].
  • SETTING: Burn Center, CHU Saint-Antoine, Paris, France. DESIGN: 130 consecutively admitted burn patients were bronchoscoped on admission [25].
  • While only 23% of admissions to the burn center were related to flame burns, these injuries accounted for 82% of children who had ARF [26].

Analytical, diagnostic and therapeutic context of Burn Units


  1. Retrospective analysis of plasmid patterns in a study of burn unit outbreaks of infection due to Enterobacter cloacae. Markowitz, S.M., Smith, S.M., Williams, D.S. J. Infect. Dis. (1983) [Pubmed]
  2. An outbreak of infections caused by strains of Staphylococcus aureus resistant to methicillin and aminoglycosides. I. Clinical studies. Crossley, K., Loesch, D., Landesman, B., Mead, K., Chern, M., Strate, R. J. Infect. Dis. (1979) [Pubmed]
  3. Environmental study of a methicillin-resistant Staphylococcus aureus epidemic in a burn unit. Rutala, W.A., Katz, E.B., Sherertz, R.J., Sarubbi, F.A. J. Clin. Microbiol. (1983) [Pubmed]
  4. Fungemia and colonization with nystatin-resistant Candida rugosa in a burn unit. Dubé, M.P., Heseltine, P.N., Rinaldi, M.G., Evans, S., Zawacki, B. Clin. Infect. Dis. (1994) [Pubmed]
  5. Comparison of polymerase chain reaction and pulsed-field gel electrophoresis for the epidemiological typing of Alcaligenes xylosoxidans subsp. xylosoxidans in a burn unit. Lin, Y.H., Liu, P.Y., Shi, Z.Y., Lau, Y.J., Hu, B.S. Diagn. Microbiol. Infect. Dis. (1997) [Pubmed]
  6. Value of molecular epidemiologic analysis in a nosocomial methicillin-resistant Staphylococcus aureus outbreak. Fang, F.C., McClelland, M., Guiney, D.G., Jackson, M.M., Hartstein, A.I., Morthland, V.H., Davis, C.E., McPherson, D.C., Welsh, J. JAMA (1993) [Pubmed]
  7. Burn units as a source of methicillin-resistant Staphylococcus aureus infections. Boyce, J.M., White, R.L., Causey, W.A., Lockwood, W.R. JAMA (1983) [Pubmed]
  8. Lorazepam as an adjunct to opioid analgesics in the treatment of burn pain. Patterson, D.R., Ptacek, J.T., Carrougher, G.J., Sharar, S.R. Pain (1997) [Pubmed]
  9. Emergence in a burn center of populations of bacteria resistant to gentamicin, tobramycin, and amikacin: evidence for the need for changes in zone diameter interpretative standards. Minshew, B.H., Pollock, H.M., Schoenknecht, F.D., Sherris, J.C. Antimicrob. Agents Chemother. (1977) [Pubmed]
  10. Ornithine alpha-ketoglutarate improves wound healing in severe burn patients: a prospective randomized double-blind trial versus isonitrogenous controls. Coudray-Lucas, C., Le Bever, H., Cynober, L., De Bandt, J.P., Carsin, H. Crit. Care Med. (2000) [Pubmed]
  11. In vitro activity of 15 antimicrobial agents against clinical isolates of Clostridium difficile in Kuwait. Jamal, W.Y., Mokaddas, E.M., Verghese, T.L., Rotimi, V.O. Int. J. Antimicrob. Agents (2002) [Pubmed]
  12. In vitro activity of piperacillin/tazobactam versus other broad-spectrum antibiotics against nosocomial gram-negative pathogens isolated from burn patients. Mokaddas, E., Rotimi, V.O., Sanyal, S.C. Journal of chemotherapy (Florence, Italy) (1998) [Pubmed]
  13. The tegument resulting from the healing of burns. Mariani, U., Gomez, D.d.e. .S., Carvalho, D.d.o. .A., Ferreira, M.C. Revista do Hospital das Clínicas. (1995) [Pubmed]
  14. Smoke inhalation and airway management at a regional burn unit: 1974-1983. Part I: Diagnosis and consequences of smoke inhalation. Clark, W.R., Bonaventura, M., Myers, W. The Journal of burn care & rehabilitation. (1989) [Pubmed]
  15. Treatment issues in the care of patients with toxic epidermal necrolysis. Smoot, E.C. Burns : journal of the International Society for Burn Injuries. (1999) [Pubmed]
  16. Methamphetamine-associated burn injuries: a retrospective analysis. Danks, R.R., Wibbenmeyer, L.A., Faucher, L.D., Sihler, K.C., Kealey, G.P., Chang, P., Amelon, M., Lewis, R.W. The Journal of burn care & rehabilitation. (2004) [Pubmed]
  17. Modulation of inflammatory and catabolic responses in severely burned children by early burn wound excision in the first 24 hours. Barret, J.P., Herndon, D.N. Archives of surgery (Chicago, Ill. : 1960) (2003) [Pubmed]
  18. Comparison of susceptibility to beta-lactam antimicrobial agents among bacteria isolated from intensive care units. Pierson, C.L., Friedman, B.A. Diagn. Microbiol. Infect. Dis. (1992) [Pubmed]
  19. Mechanisms of in vitro sensitivity to sulfadiazine silver. McManus, A.T., Denton, C.L., Mason, A.D. Archives of surgery (Chicago, Ill. : 1960) (1983) [Pubmed]
  20. Burn center care for patients with toxic epidermal necrolysis. Kelemen, J.J., Cioffi, W.G., McManus, W.F., Mason, A.D., Pruitt, B.A. J. Am. Coll. Surg. (1995) [Pubmed]
  21. Facial and ocular injuries associated with methamphetamine production accidents. Charukamnoetkanok, P., Wagoner, M.D. Am. J. Ophthalmol. (2004) [Pubmed]
  22. Staphylococcal septicaemia in burns. Gang, R.K., Sanyal, S.C., Bang, R.L., Mokaddas, E., Lari, A.R. Burns : journal of the International Society for Burn Injuries. (2000) [Pubmed]
  23. Evolution and significance of circulating procalcitonin levels compared with IL-6, TNF alpha and endotoxin levels early after thermal injury. Carsin, H., Assicot, M., Feger, F., Roy, O., Pennacino, I., Le Bever, H., Ainaud, P., Bohuon, C. Burns : journal of the International Society for Burn Injuries. (1997) [Pubmed]
  24. An outbreak of infections caused by strains of Staphylococcus aureus resistant to methicillin and aminoglycosides. II. Epidemiologic studies. Crossley, K., Landesman, B., Zaske, D. J. Infect. Dis. (1979) [Pubmed]
  25. Using bronchoscopy and biopsy to diagnose early inhalation injury. Macroscopic and histologic findings. Masanès, M.J., Legendre, C., Lioret, N., Saizy, R., Lebeau, B. Chest (1995) [Pubmed]
  26. Mortality and respiratory failure in a pediatric burn population. Reynolds, E.M., Ryan, D.P., Doody, D.P. J. Pediatr. Surg. (1993) [Pubmed]
  27. The antifibrinolytic activity of sulfamylon solution. Aldridge, J.H., Weisdorf, D.J., Kucan, J.O. Plast. Reconstr. Surg. (1988) [Pubmed]
  28. Plasma fibronectin time course in burned patients: influence of sepsis. Ekindjian, O.G., Marien, M., Wassermann, D., Bruxelle, J., Cazalet, C., Konter, E., Yonger, J. The Journal of trauma. (1984) [Pubmed]
  29. An outbreak of methicillin resistant Staphylococcus aureus on a burn unit: potential role of contaminated hydrotherapy equipment. Embil, J.M., McLeod, J.A., Al-Barrak, A.M., Thompson, G.M., Aoki, F.Y., Witwicki, E.J., Stranc, M.F., Kabani, A.M., Nicoll, D.R., Nicolle, L.E. Burns : journal of the International Society for Burn Injuries. (2001) [Pubmed]
  30. Hyperbaric oxygen in the treatment of burns. Grossman, A.R. Annals of plastic surgery. (1978) [Pubmed]
WikiGenes - Universities