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MeSH Review

Residual Volume

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Disease relevance of Residual Volume


High impact information on Residual Volume


Chemical compound and disease context of Residual Volume


Biological context of Residual Volume


Anatomical context of Residual Volume


Associations of Residual Volume with chemical compounds

  • In 6 normal subjects given erythromycin three times weekly for 1 month, the effect was maintained (fasting volume, 18.8 mL, P less than 0.001; residual volume, 3.7 mL, P less than 0.001) [19].
  • By 6 months, mean urinary flow rates had increased (p less than 0.05) and residual volume had decreased (p = 0.017) in the alfuzosin group, although the two groups were broadly similar with respect to increase in peak flow rate [20].
  • In this heavily pretreated patient population, most of whom had high volume residual disease, complete responses (CRs) to CBV and ABMT occurred in 25% of assessable patients with NHL and 43% of patients with HD [21].
  • Treatment with octreotide increased the mean postprandial residual volume further to 36.8 ml (p < 0.001) [22].
  • Fasting gall bladder volume was increased during simvastatin (28.7 (2.8) ml) v baseline (23.2 (2.3) ml, p < 0.01) whereas, residual volume did not differ (5.7 (0.9) ml (simvastatin) v 5.9 (0.7) (baseline) [23].

Gene context of Residual Volume


Analytical, diagnostic and therapeutic context of Residual Volume


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