Disease relevance of Thrombophlebitis

• Among all 40 patients treated with MGDF, 1 had deep venous thrombosis and pulmonary embolism, and another had superficial thrombophlebitis [1].
• Neither IVCT nor RVT was found in SLE patients without antecedent thrombophlebitis or nephrotic syndrome [2].
• Thirty-day mortality rates for several complications exceeded 50%: cardiac arrest requiring cardiopulmonary resuscitation, deep venous thrombosis or thrombophlebitis, coma lasting >24 hours, acute renal failure, cerebrovascular accident, and pulmonary embolism [3].
• Except for transient elevation in transaminases in one patient and occasional infusion thrombophlebitis, no toxicity of acyclovir was encountered in this series [4].
• Serious medical problems coexisted, and all patients had at least two of five important "risk factors." These were heart disease, recent thrombophlebitis, heparin therapy, abdominal atherosclerosis and postoperative status [5].

Psychiatry related information on Thrombophlebitis

• Midazolam was associated with better patient tolerance, less thrombophlebitis, and more amnesia than diazepam, but shared a similar recovery time [6].
• Deep venous thrombosis and venous thrombophlebitis associated with alprostadil treatment for erectile dysfunction [7].

High impact information on Thrombophlebitis

• Two cases of cutaneous necrosis occurred that were caused by intravenous administration of porcine heparin for deep venous thrombophlebitis [8].
• Thrombophlebitis in systemic lupus erythematosus [9].
• Four further patients who wore elastic compression stockings, had varicose veins and developed superficial thrombophlebitis [10].
• In the azathioprine group, the only thromboembolic complication was 1 episode of superficial thrombophlebitis [11].
• Ketanserin in acute superficial thrombophlebitis [12].

Chemical compound and disease context of Thrombophlebitis

• Vitamin E and thrombophlebitis [13].
• PURPOSE: To determine the role of the fluorine-18 fluorodeoxyglucose positron emission tomography (FDG-PET) scan in the diagnosis and management of deep septic thrombophlebitis (STP) [14].
• PURPOSE: Estrogen replacement therapy is believed by many physicians to cause thrombophlebitis and to be contraindicated in women at risk for this disease [15].
• Venous thrombophlebitis may complicate tamoxifen treatment at a rate of approximately one per 800 treatment-years [16].
• Minor bleeding and thrombophlebitis occurred more frequently in the efegatran treated patients [17].

Biological context of Thrombophlebitis

• Although pain and subsequent thrombophlebitis are complications in patients receiving intravenous (IV) diazepam, the mechanism and accompanying histology are unknown [18].
• The diagnosis was biological on low platelet counts (p.c.) in 4 cases, in 7 cases a deep venous thrombophlebitis and in 9 cases an acute arterial ischemia complicated the heparin treatment [19].
• A 60-year-old white male on warfarin therapy for recurrent pulmonary embolism and deep venous thrombophlebitis showed a decrease in his previously therapeutic and stable prothrombin time when piroxicam was discontinued from his drug regimen [20].

Anatomical context of Thrombophlebitis

• In a double-blind study, the efficacy and tolerability of nimesulide (100mg twice daily) were compared with those of diclofenac (50mg twice daily) when administered orally to 50 patients with acute superficial thrombophlebitis of the lower limbs [21].
• On the basis of clinical experience it has been claimed that glucose results in a higher incidence of infusion thrombophlebitis than fructose, and that glucose solutions in concentrations higher than 5% are extremely irritant to the venous endothelium [22].
• Cavernous sinus thrombophlebitis in Nijmegen breakage syndrome [23].
• Bilateral upper extremity thrombophlebitis related to intravenous amiodarone: a case report [24].
• In patients with thrombophlebitis receiving intravenously a nicotinic acid derivative, pentoxifylline and streptokinase the fibrinolytic activity and leukocyte adhesiveness were studied [25].

Gene context of Thrombophlebitis

• The clinical features of thromboembolism were similar in the three deficiency states except for a higher frequency of superficial thrombophlebitis in patients with PC and PS deficiencies [26].
• Thrombophlebitis of superficial veins was found to be very common in patients with protein C and protein S deficiency and virtually absent in AT-III deficiency [27].
• The aim of this study was to investigate the interactions of t-PA and plasminogen with fibrin derived from an abnormal fibrinogen detected in a 40-year-old male patient who had had an episode of thrombophlebitis with pulmonary embolism [28].
• His mother has protein C deficiency and showed a single episode of thrombophlebitis following surgery [29].
• BACKGROUND: The contribution of glyceryl trinitrate (GTN) to prevention of peripheral vein thrombophlebitis (PVT) during peripheral intravenous nutrition delivered by fine-bore midline intravenous catheter is unclear [30].

Analytical, diagnostic and therapeutic context of Thrombophlebitis

• Contrast agent-induced thrombophlebitis following leg phlebography: meglumine loxaglate versus meglumine lothalamate [31].
• Detection of deep venous thrombophlebitis by gallium 67 scintigraphy [32].
• The incidence of thrombophlebitis was then determined using the iodine-125 fibrinogen uptake test in a prospective, randomized, double-blind study [31].
• Next, thrombophlebitis was produced in dogs by a single intravenous injection of 1 ml 90% phenol into a leg vein occluded for 1 min by a gauze tourniquet and the effects of thrombophlebitis were studied [33].
• Long-term, intermittent, intravenous infusion of substances, such as amphotericin B, with a high potential for causing peripheral vein thrombophlebitis was feasible by means of a subcutaneously implanted silastic reservoir catheter device, which gave access to the central venous compartment [34].

References