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MeSH Review

Ovarian Diseases

 
 
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Disease relevance of Ovarian Diseases

 

High impact information on Ovarian Diseases

 

Chemical compound and disease context of Ovarian Diseases

 

Biological context of Ovarian Diseases

  • Successful induction of ovulation and conception with combined gonadotropin-releasing hormone agonist plus highly purified follicle-stimulating hormone in patients with polycystic ovarian disease [16].
  • PCA analysis allowed correct separation of all serum specimens from 38 patients with EOC (100%) from all of the 21 premenopausal normal samples (100%) and from all the sera from patients with benign ovarian disease (100%) [17].
  • Administration of 4 mg of the antiprogestagen RU486 to 4-day-cyclic rats over 8 consecutive days starting on the day of estrus (Day 1) induced and anovulatory cystic ovarian condition with endocrine and morphological features similar to those exhibited in polycystic ovarian disease (PCO) [18].
  • To evaluate the role of altered luteinizing hormone (LH) release in the mechanism of polycystic ovarian disease (PCOD) anovulation, we have co-administered a gonadotrophin-releasing hormone (GnRH) antagonist and pulsatile GnRH therapy to two clomiphene citrate-resistant PCOD patients [19].
  • Serum gonadotropin, estrogen, and androgen levels were measured in samples obtained from 19 patients with polycystic ovarian disease (PCO) and from 10 normal women on day 2 to 4 of their menstrual cycles [20].
 

Anatomical context of Ovarian Diseases

 

Gene context of Ovarian Diseases

 

Analytical, diagnostic and therapeutic context of Ovarian Diseases

References

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  18. RU486-treated rats show endocrine and morphological responses to therapies analogous to responses of women with polycystic ovary syndrome treated with similar therapies. Ruiz, A., Aguilar, R., Tébar, A.M., Gaytán, F., Sánchez-Criado, J.E. Biol. Reprod. (1996) [Pubmed]
  19. The combination of gonadotrophin-releasing hormone (GnRH) antagonist and pulsatile GnRH normalizes luteinizing hormone secretion in polycystic ovarian disease but fails to induce follicular maturation. Dubourdieu, S., Le Nestour, E., Spitz, I.M., Charbonnel, B., Bouchard, P. Hum. Reprod. (1993) [Pubmed]
  20. Circulating gonadotropins, estrogens, and androgens in polycystic ovarian disease. DeVane, G.W., Czekala, N.M., Judd, H.L., Yen, S.S. Am. J. Obstet. Gynecol. (1975) [Pubmed]
  21. Luteinizing hormone receptors in ovarian follicles of patients with polycystic ovarian disease. Rajaniemi, H.J., Rönnberg, L., Kauppila, A., Ylöstalo, P., Vihko, R. J. Clin. Endocrinol. Metab. (1980) [Pubmed]
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