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MeSH Review

Extracorporeal Membrane Oxygenation

 
 
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Disease relevance of Extracorporeal Membrane Oxygenation

 

High impact information on Extracorporeal Membrane Oxygenation

 

Chemical compound and disease context of Extracorporeal Membrane Oxygenation

 

Biological context of Extracorporeal Membrane Oxygenation

 

Anatomical context of Extracorporeal Membrane Oxygenation

 

Associations of Extracorporeal Membrane Oxygenation with chemical compounds

 

Gene context of Extracorporeal Membrane Oxygenation

  • We investigated the expression pattern of NO-generating enzyme nitric-oxide synthase (NOS) (both endothelial [eNOS] and inducible [iNOS] isoforms) in the lungs of CDH patients with PH and evaluated the influence of ECMO on the expression levels of these genes in an attempt to understand the underlying molecular mechanisms [30].
  • CONCLUSIONS: These data suggest that the infants who survive ECMO resolve their pulmonary inflammation and that in non-survivors the ratio of TNF-alpha against its receptor antagonists is increased and is associated with a poor outcome [31].
  • Concentrations of circulating neutrophil elastase increase significantly during ECMO [32].
  • Plasma from the ECMO experiments did not induce ICAM-1 expression of cultured HUVEC [33].
  • MMP-2 may play a role in the development of platelet dysfunction caused by ECMO [34].
 

Analytical, diagnostic and therapeutic context of Extracorporeal Membrane Oxygenation

  • The data from this report support the concept that infants undergoing surgical repair of diaphragmatic hernia, when ECMO is not available, should be referred to an ECMO center in the early postoperative period [35].
  • Furthermore infants with prenatal diagnosis of diaphragmatic hernia should be delivered at a center where surgical as well as ECMO expertise are available [35].
  • Concomitant procedures were frequently performed and included repair of an intra-atrial baffle leak (prior Mustard procedure), closure of an atrial septal defect, repair of partial anomalous pulmonary venous return, reconstruction of the pulmonary venous confluence, ECMO decannulation, and splenectomy [36].
  • Fourteen did not receive allopurinol, whereas 11 were treated with 10 mg/kg after meeting criteria and before cannulation, in addition to a 20-mg/kg priming dose to the ECMO circuit [37].
  • This amount of DEHP extrapolates in the ECMO patient to a potential exposure of 20 to 70 times that exposure from other medical devices or procedures, such as transfusions, dialysis, or short-term cardiopulmonary bypass [13].

References

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  22. Nucleated erythrocyte count in newborn infants with left-sided congenital diaphragmatic hernia: relationship with the need for extracorporeal membrane oxygenation and survival. Green, D.W., Lyon, J., Ackerman, N.B., Mimouni, F. J. Pediatr. (1995) [Pubmed]
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  24. Congenital diaphragmatic hernia without herniation of the liver: does the lung-to-head ratio predict survival? Sbragia, L., Paek, B.W., Filly, R.A., Harrison, M.R., Farrell, J.A., Farmer, D.L., Albanese, C.T. Journal of ultrasound in medicine : official journal of the American Institute of Ultrasound in Medicine. (2000) [Pubmed]
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  29. Neonates with congenital diaphragmatic hernia: radiographic findings during partial liquid ventilation. Garver, K.A., Kazerooni, E.A., Hirschl, R.B., DiPietro, M.A. Radiology. (1996) [Pubmed]
  30. Pulmonary hypertension in human newborns with congenital diaphragmatic hernia is associated with decreased vascular expression of nitric-oxide synthase. Shehata, S.M., Sharma, H.S., Mooi, W.J., Tibboel, D. Cell Biochem. Biophys. (2006) [Pubmed]
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  33. Leukocyte and endothelial activation in a laboratory model of extracorporeal membrane oxygenation (ECMO). Graulich, J., Walzog, B., Marcinkowski, M., Bauer, K., Kössel, H., Fuhrmann, G., Bührer, C., Gaehtgens, P., Versmold, H.T. Pediatr. Res. (2000) [Pubmed]
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