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MeSH Review

Brain Infarction

 
 
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Disease relevance of Brain Infarction

 

Psychiatry related information on Brain Infarction

 

High impact information on Brain Infarction

 

Chemical compound and disease context of Brain Infarction

 

Biological context of Brain Infarction

 

Anatomical context of Brain Infarction

 

Gene context of Brain Infarction

  • To better understand the interaction of pro- and antiinflammatory acute phase processes in brain infarction, plasma levels of IL-1RA, IL-6, and acute phase proteins including fibrinogen and c-reactive protein (CRP) were measured within 4 +/- 2 days of onset in 50 patients with acute ischemic stroke and in 20 age-matched healthy controls [27].
  • The ischaemic leukoaraiosis group had a different endothelial marker profile, with lower levels of TFPI (P = 0.01) and a higher TF/TFPI ratio (P = 0.01) compared with the isolated lacunar infarction group [28].
  • BACKGROUND AND PURPOSE: We initiated the present study to evaluate the clinical value of consecutive concentration determinations of S-100 and glial fibrillary acidic proteins in cerebrospinal fluid from patients with brain infarction [29].
  • The highest CA II concentrations were found in patients with brain infarction (median 66.5 micrograms L-1, n = 20), whereas the control patients had markedly lower values (median 7.8 micrograms L-1, n = 97) [30].
  • Well-characterized antisera were therefore used to examine the neuronal expression of CDK2, CDK4 and cyclins A, D1 and E in sections of brain from patients who had experienced cardiac arrest or focal brain infarction, and died 3.5 h to 9 days later [31].
  • Our results indicate that the SNP in the AGTRL1 gene is associated with the susceptibility to brain infarction [32].
 

Analytical, diagnostic and therapeutic context of Brain Infarction

References

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  2. Homocysteine is a risk factor for cerebral small vessel disease, acting via endothelial dysfunction. Hassan, A., Hunt, B.J., O'Sullivan, M., Bell, R., D'Souza, R., Jeffery, S., Bamford, J.M., Markus, H.S. Brain (2004) [Pubmed]
  3. Automated measurement of infarct size with scanned images of triphenyltetrazolium chloride-stained rat brains. Goldlust, E.J., Paczynski, R.P., He, Y.Y., Hsu, C.Y., Goldberg, M.P. Stroke (1996) [Pubmed]
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  17. Impairments of the protein C system and fibrinolysis in infection-associated stroke. Macko, R.F., Ameriso, S.F., Gruber, A., Griffin, J.H., Fernandez, J.A., Barndt, R., Quismorio, F.P., Weiner, J.M., Fisher, M. Stroke (1996) [Pubmed]
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  19. The transcription factor E2F1 modulates apoptosis of neurons. Hou, S.T., Callaghan, D., Fournier, M.C., Hill, I., Kang, L., Massie, B., Morley, P., Murray, C., Rasquinha, I., Slack, R., MacManus, J.P. J. Neurochem. (2000) [Pubmed]
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  22. Allopurinol and dimethylthiourea reduce brain infarction following middle cerebral artery occlusion in rats. Martz, D., Rayos, G., Schielke, G.P., Betz, A.L. Stroke (1989) [Pubmed]
  23. Imaging of leukocytic infiltration in human cerebral infarcts. Pozzilli, C., Lenzi, G.L., Argentino, C., Carolei, A., Rasura, M., Signore, A., Bozzao, L., Pozzilli, P. Stroke (1985) [Pubmed]
  24. Neuropsychological correlates of a right unilateral lacunar thalamic infarction. Van Der Werf, Y.D., Weerts, J.G., Jolles, J., Witter, M.P., Lindeboom, J., Scheltens, P. J. Neurol. Neurosurg. Psychiatr. (1999) [Pubmed]
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  29. Determination of S-100 and glial fibrillary acidic protein concentrations in cerebrospinal fluid after brain infarction. Aurell, A., Rosengren, L.E., Karlsson, B., Olsson, J.E., Zbornikova, V., Haglid, K.G. Stroke (1991) [Pubmed]
  30. Carbonic anhydrase II in the cerebrospinal fluid: its value as a disease marker. Parkkila, A.K., Parkkila, S., Reunanen, M., Niemelä, O., Tuisku, S., Rautakorpi, I., Rajaniemi, H. Eur. J. Clin. Invest. (1997) [Pubmed]
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  32. Functional SNP in an Sp1-binding site of AGTRL1 gene is associated with susceptibility to brain infarction. Hata, J., Matsuda, K., Ninomiya, T., Yonemoto, K., Matsushita, T., Ohnishi, Y., Saito, S., Kitazono, T., Ibayashi, S., Iida, M., Kiyohara, Y., Nakamura, Y., Kubo, M. Hum. Mol. Genet. (2007) [Pubmed]
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  36. Association studies between haemochromatosis gene mutations and the risk of cardiovascular diseases. Hetet, G., Elbaz, A., Gariepy, J., Nicaud, V., Arveiler, D., Morrison, C., Kee, F., Evans, A., Simon, A., Amarenco, P., Cambien, F., Grandchamp, B. Eur. J. Clin. Invest. (2001) [Pubmed]
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