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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 
MeSH Review

Candidiasis

 
 
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Disease relevance of Candidiasis

 

Psychiatry related information on Candidiasis

 

High impact information on Candidiasis

 

Chemical compound and disease context of Candidiasis

 

Biological context of Candidiasis

 

Anatomical context of Candidiasis

 

Gene context of Candidiasis

  • Therapeutic ablation of IL-4 or IL-10 greatly increased resistance to candidiasis [28].
  • Taken together, our findings suggest that SET1 regulates multiple processes important to the pathogenesis of candidiasis [29].
  • Despite the importance of interferon (IFN)-gamma, tumor necrosis factor (TNF), and interleukin (IL)-18 for host defense against candidiasis, the pathways leading to their stimulation by Candida albicans are unclear [30].
  • The secreted aspartyl proteinases Sap1 and Sap2 cause tissue damage in an in vitro model of vaginal candidiasis based on reconstituted human vaginal epithelium [31].
  • We implicate C. albicans NOT5 in the pathogenesis of disseminated candidiasis (DC) induced by intravenous infection among neutropenic and nonimmunosuppressed mice, as well as in that of OPC in mice immunosuppressed with corticosteroids [32].
 

Analytical, diagnostic and therapeutic context of Candidiasis

References

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