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MeSH Review

Cerebellar Nuclei

 
 
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Disease relevance of Cerebellar Nuclei

 

Psychiatry related information on Cerebellar Nuclei

 

High impact information on Cerebellar Nuclei

  • At stages prior to E12.5, with the exception of the deep cerebellar nuclei, we find that Math1 cells migrate out of the cerebellar primordium into the rostral hindbrain to populate specific nuclei that include cholinergic neurons of the mesopontine tegmental system [8].
  • Cerebellar ataxia, a devastating neurological disease, may be initiated by hyperexcitability of deep cerebellar nuclei (DCN) secondary to loss of inhibitory input from Purkinje neurons that frequently degenerate in this disease [9].
  • In cryostat sections, neurons that express proenkephalin include Golgi cells, macroneurons within deep cerebellar nuclei and a subpopulation of Purkinje cells [10].
  • Netrin-G1 transcripts were first detected in midbrain and hindbrain regions by embryonic day 12 and reached highest levels at perinatal stages in various brain regions, including olfactory bulb mitral cells, thalamus, and deep cerebellar nuclei [11].
  • In all 7 cases, elevated serotonin synthesis in the contralateral dentate nucleus was observed [12].
 

Chemical compound and disease context of Cerebellar Nuclei

 

Biological context of Cerebellar Nuclei

 

Anatomical context of Cerebellar Nuclei

 

Associations of Cerebellar Nuclei with chemical compounds

 

Gene context of Cerebellar Nuclei

 

Analytical, diagnostic and therapeutic context of Cerebellar Nuclei

References

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