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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 
MeSH Review

Reperfusion

 
 
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Disease relevance of Reperfusion

 

Psychiatry related information on Reperfusion

 

High impact information on Reperfusion

 

Chemical compound and disease context of Reperfusion

 

Biological context of Reperfusion

 

Anatomical context of Reperfusion

 

Associations of Reperfusion with chemical compounds

  • We found that mice homozygous deficient in C3 or C4 were equally protected against reperfusion injury based on a significant reduction in leakage of radiolabeled albumin out of the vasculature [27].
  • Specifically, promethazine protected primary neuronal cultures subjected to oxygen-glucose deprivation and reduced infarct size and neurological impairment in mice subjected to middle cerebral artery occlusion/reperfusion [28].
  • Creatine kinase, an indicator of cellular injury, was released at a high level (67.7 +/- 23.0 U/ml) upon reperfusion from nontransgenic hearts, but not transgenic hearts (1.6 +/- 0.8 U/ml) [29].
  • Unlike the effect on tissue MDA and conjugated dienes, reperfusion did not significantly stimulate release of MDA in the cardiac effluent [30].
  • During reperfusion, the sinusoidal efflux of total glutathione (16.4 +/- 2.1 nmol GSH eq/min X g) and GSSG (0.13 +/- 0.05 nmol GSH eq/min X g) did not change except for an initial 10-30-s increase during reperfusion washout [31].
 

Gene context of Reperfusion

 

Analytical, diagnostic and therapeutic context of Reperfusion

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