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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 
MeSH Review

Withholding Treatment

 
 
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Disease relevance of Withholding Treatment

 

Psychiatry related information on Withholding Treatment

 

High impact information on Withholding Treatment

  • Six months after the cessation of treatment, plasma testosterone levels had returned to normal and the size of the prostate had increased to 99 +/- 5.5 percent of the initial size [11].
  • BAY 57-1293 (N-[5-(aminosulfonyl)-4-methyl-1,3-thiazol-2-yl]-N-methyl-2-[4-(2-pyridinyl)phenyl]acetamide), a well-tolerated member of this class of compounds, significantly reduces time to healing, prevents rebound of disease after cessation of treatment and, most importantly, reduces frequency and severity of recurrent disease [12].
  • Cessation of treatment and recovery allowed us to monitor reemergence of these cell types and revealed that FDCs rely on LT-dependent signals to recompact into appropriately positioned tight networks [13].
  • After cessation of treatment with mexiletine the number of sodium channels returned to normal within 12 d [14].
  • Following cessation of treatment, analog II prevented metastatic tumor development for up to a month and tamoxifen, for 3 weeks [15].
 

Chemical compound and disease context of Withholding Treatment

 

Biological context of Withholding Treatment

 

Anatomical context of Withholding Treatment

 

Associations of Withholding Treatment with chemical compounds

 

Gene context of Withholding Treatment

  • Animals given IL-6 or IL-3 plus IL-6 were subsequently thrombocytopenic relative to controls on days 7 through 9 following cessation of treatment [36].
  • The suppressive effect of IL-3 was evident throughout the treatment period (8 or 16 days) but was apparently reversible because NK activity returned to control levels within 8 days after cessation of treatment [37].
  • The eight tumors that regressed with the liposome-p53 complex showed no evidence of relapse for 1 month after the cessation of treatment [38].
  • BDNF-induced survival of auditory neurons in vivo: Cessation of treatment leads to accelerated loss of survival effects [39].
  • In gastrin-deficient mice, significant SPEM was still present 14 days after the cessation of treatment [40].
 

Analytical, diagnostic and therapeutic context of Withholding Treatment

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