Disease relevance of Enteritis

• Neurokinin-1 (NK-1) receptor is required in Clostridium difficile- induced enteritis [1].
• Pancreatitis associated with Salmonella enteritis [2].
• The ability of endogenous breastmilk xanthine oxidase to generate the antimicrobial radical nitric oxide has been measured and its influence on the growth of Escherichia coli and Salmonella enteritides examined [3].
• L-glutamine stimulates jejunal sodium and chloride absorption in pig rotavirus enteritis [4].
• Reactive arthritis associated with Hafnia alvei enteritis [5].

High impact information on Enteritis

• We conclude that in this invasive enteritis caused by a major human viral pathogen, glucose-coupled Na+ transport is impaired in the jejunum at a time when the villus epithelium shows enzyme characteristics of crypt epithelium, and when little or no virus is present [6].
• There were significantly fewer admissions for septicaemia and enteritis in the co-trimoxazole group than in the placebo group [7].
• Treatment of gold-induced enteritis with octreotide [8].
• Campylobacter enteritis during doxycycline prophylaxis for malaria in Thailand [9].
• Double-blind placebo controlled trial of erythromycin for treatment of Campylobacter enteritis [10].

Chemical compound and disease context of Enteritis

• Ulcerating enteritis associated with flucytosine therapy [11].
• Ten patients with radiation enteritis unresponsive to conventional medical and surgical therapy were put on long-term parenteral nutrition at home [12].
• Two patients in our series were given prednisone and slinical improvement of radiation enteritis with this therapy [12].
• Administration of a chemically defined liquid diet to rats receiving methotrexate increased the occurrence and severity of intestinal enteritis, altered intraluminal bowel flora, and decreased clearance of methotrexate from the serum [13].
• Development of multiple necrotizing enteritis induced by a tumor necrosis factor-like cytokine from lipopolysaccharide-stimulated peritoneal macrophages in rats [14].

Biological context of Enteritis

• Inflammatory bowel disease-like enteritis and caecitis in a senescence accelerated mouse P1/Yit strain [15].
• Kinetics of anti-Campylobacter jejuni monomeric and polymeric immunoglobulin A1 and A2 responses in serum during acute enteritis [16].
• By use of reverse-transcription polymerase chain reaction to detect the viral RNA polymerase gene in stool and tissue from gastrointestinal biopsies, 5 pediatric IT recipients with high-volume diarrhea were diagnosed with HuCV enteritis [17].
• Genotype analysis of two of these isolates revealed them to be C. perfringens type A. Bacterial enteritis is believed to be the cause of the increased mortality rate, but further investigation is required to prove a definitive link to a clostridial agent [18].
• As the amount of cortisone given to mice was increased enteritis decreased, and fecundity of adult worms increased [19].

Anatomical context of Enteritis

• Histamine, mast cells, and the enteric nervous system in the irritable bowel syndrome, enteritis, and food allergies [20].
• A Campylobacter-like organism, BM2196, resistant to kanamycin and streptomycin-spectinomycin was isolated from the feces of a patient with acute enteritis [21].
• We sought to determine the activity of lactoferrin in vivo, using the concentration present in human colostrum, to investigate its effect on the development of clinical and pathological evidence of inflammation in a rabbit model of enteritis [22].
• Our findings support the concept that, in viral enteritis, oral rehydration solutions containing amino acid and glucose have a theoretical advantage over glucose electrolyte solutions because they facilitate brush border Na+ entry by two carrier mechanisms [23].
• In addition to our previous studies and results in indomethacin induced enteritis, these data may provide a rationale for studying how UDCA modulates functions of immune cells in the colonic mucosa [24].

Gene context of Enteritis

• In this study, we assessed whether B. fragilis enterotoxin (BFT)-induced enteritis is related to mitogen-activated protein kinase (MAPK) signaling pathways [25].
• Mitogen-activated protein kinase and activator protein-1 dependent signals are essential for Bacteroides fragilis enterotoxin-induced enteritis [25].
• CTGF immunoreactivity, protein, and mRNA level were increased in radiation enteritis compared with the healthy bowel [26].
• We determined whether the SP-degrading enzyme neutral endopeptidase (NEP, EC 3.4.24.11) terminates TxA-induced enteritis [27].
• In the present study, circulating IGF-I, IGF-II and their binding proteins (IGFBP-3, IGFBP-2 and IGFBP-1) were determined in 92 adult patients with gastrointestinal inflammation (Crohn's disease, colitis ulcerosa, gastritis, duodenitis errosiva, gastrointestinal candidiasis, and rotaviral and adenoviral enteritis) [28].

Analytical, diagnostic and therapeutic context of Enteritis

• Two of 54 patients with Campylobacter enteritis had a clinical relapse after treatment that was associated with development of ciprofloxacin resistance [29].
• To prevent or minimize mizoribine enterotoxicity in organ transplant recipients and to differentiate mizoribine enterotoxicity from other causes of enteritis, serum levels of mizoribine that produced subclinical and clinical signs of enterotoxicity were determined in the dog [30].
• The in vitro susceptibilities suggest that clinical trials to treat enteritis caused by multidrug-resistant C. jejuni with co-amoxiclav, and life-threatening infections with a carbapenem, may be valuable [31].
• Late complications included life-threatening events: Six episodes of gastro intestinal bleeding (three of them had an arteriographic documentation of arterioenteric fistula) and nine with severe enteritis (five required reoperation) [32].
• Methicillin-resistant Staphylococcus aureus (MRSA) enteritis associated with prophylactic cephalosporin administration and hypochlorhydria, after subtotal gastrectomy [33].

References