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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 
MeSH Review

Tissue Survival

 
 
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Disease relevance of Tissue Survival

 

High impact information on Tissue Survival

 

Chemical compound and disease context of Tissue Survival

 

Biological context of Tissue Survival

 

Anatomical context of Tissue Survival

 

Associations of Tissue Survival with chemical compounds

 

Gene context of Tissue Survival

  • The increased tissue survival associated with the capacity for iNOS expression may be related to iNOS-dependent enhancement of VEGF levels and an ensuing angiogenic response [30].
  • Thyroid tissue viability was maintained or increased by antigen-specific Th0 clones, and decreased by Th1 clones reactive to TSH-R or TPO [31].
  • The results of the present study suggest that higher concentrations of CGRP-LI are related to tissue survival and that endogenous CGRP has a regulatory effect in ischaemia [32].
  • Depolarizing concentrations of potassium (40 mM) added to medium after incubation in the presence or absence of glucose produced similar 4-fold biphasic stimulation of SRIF release, suggesting that tissue viability was unimpaired by short term glucopenia [33].
  • The perfusion of severed limbs and digits and free vascularized tissue transfers with superoxide dismutase and catalase after a period of ischemia has already occurred may prolong the ischemic "time window" tolerated for successful tissue survival [34].
 

Analytical, diagnostic and therapeutic context of Tissue Survival

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