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Hoffmann, R. A wiki for the life sciences where authorship matters. Nature Genetics (2008)
 
MeSH Review

Neural Pathways

 
 
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Disease relevance of Neural Pathways

 

Psychiatry related information on Neural Pathways

 

High impact information on Neural Pathways

 

Chemical compound and disease context of Neural Pathways

 

Biological context of Neural Pathways

 

Anatomical context of Neural Pathways

 

Associations of Neural Pathways with chemical compounds

  • To provide a link between the clock and the sun compass, we identified a CRYPTOCHROME-staining neural pathway that likely connects the circadian clock to polarized light input entering brain [30].
  • Pancreatic responses to intraduodenal administration of maltose, hyperosmolar NaCl, and light mucosal stroking were examined in rats pretreated with various pharmacological antagonists or after surgical or chemical ablation of vagal and 5-HT neural pathways [31].
  • CONCLUSIONS: These results indicate that intragastric capsaicin predominantly stimulates motility in the proximal half of the colon through neural pathways, probably through a vagovagal reflex, but sympathetic involvement cannot be excluded [32].
  • This effect is reversed by hexamethonium, suggesting that a neural pathway is involved [33].
  • To determine the nature of the neural pathways mediating the bombesin-induced somatostatin release, atropine (100 nmol/L) was used [34].
 

Gene context of Neural Pathways

  • The excitatory effect is also dependent on sensory neural pathways and requires both NK1 and NK2 receptors [35].
  • In conclusion, we have found that the proinflammatory cytokine IL-1beta specifically activates certain neurochemically defined neural pathways and that these changes may lead to disturbances in motility observed in the inflamed bowel [36].
  • Furthermore, the data support the notion that rolipram, the PDE4-specific inhibitor that acts as an antidepressant in humans, may mediate its behavioral effects through PDE4B, which is highly localized to neural pathways known to underlie reward and affect in mammals [37].
  • Recent studies in female mice that cannot synthesize oxytocin (OT) suggest that central OT neural pathways attenuate the response of the hypothalamic-pituitary-adrenal (HPA) axis to certain stressors [38].
  • It appears therefore that, as expected, NPY and alpha-MSH closely interact in the control of feeding, whereas the neural pathways by which NPY affects growth and reproduction are distinct and not sensitive to MC peptide modulation [39].
 

Analytical, diagnostic and therapeutic context of Neural Pathways

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